Nutritional secondary hyperparathyroidism (NSH) was diagnosed in six cats d
uring; a three-year period, based on clinical, radiographic and laboratory
findings, Clinical signs were attributable to severe osteopenia (n=5) and h
ypocalcaemia (n=4), which had resulted in spontaneous fractures of long bon
es, scapulae, pelvis, nasal bones, or spine, and in excitation, muscle twit
ching or seizures, respectively, Serum parathormone levels were markedly el
evated, and 1,25(OH)(2)-vitamin D-3 mildly elevated, whereas 25(OH)-vitamin
D-3 was mildly decreased compared to age-matched healthy cats. Treatment w
as limited to short-term parenteral calcium gluconate injections, as clinic
ally indicated, a balanced diet and cage rest, which resulted in quick clin
ical recovery in four cases, The remaining two cats had to be euthanased be
cause of progressive neurological deficits secondary to spinal fractures, A
t the time of writing, a multitude of commercial balanced diets is widely a
vailable and diseases secondary to dietary deficiencies have become rare, N
evertheless, NSH is still an important clinical entity, and should be consi
dered in growing cats presenting with spontaneous fractures or seizures.