The evaluation of the depressed newborn as a possible role in intrapartum a
sphyxia must be based on the objective findings of the fetal heart rate tra
cing, umbilical cord blood gas, and newborn neurologic function. Using thes
e points as a basis for long-term followup, the following principles have b
een elucidated: Intrapartum asphyxia is an uncommon cause of childhood neur
ologic dysfunction, the intrapartum insult necessary to cause long-term neu
rologic dysfunction is profound, and the child who does not manifest enceph
alopathy in the newborn period will not suffer long-term neurologic dysfunc
tion that can be attributed to intrapartum asphyxia.