Purpose: To demonstrate that progressive visual field loss may occur after
resolution of papilledema in patients with idiopathic intracranial hyperten
sion and persistently elevated intracranial pressure.
Methods: A patient with idiopathic intracranial hypertension was evaluated
with serial Humphrey automated static perimetry after initial treatment and
resolution of papilledema.
Results: The patient developed recurrent headache and elevated cerebrospina
l fluid pressure. Optic nerve head appearance did not change. Automated per
imetry demonstrated reproducible, worsening visual field loss; mean deviati
on decreased 11 dB in each eye. Visual field defects resolved after optic n
erve sheath fenestration.
Conclusions: Increased intracranial pressure caused visual field loss after
resolution of papilledema. Optic nerve sheath fenestration improved visual
function in this patient.