Cd. Harner et al., Biomechanical analysis of a posterior cruciate ligament reconstruction - Deficiency of the posterolateral structures as a cause of graft failure, AM J SP MED, 28(1), 2000, pp. 32-39
We hypothesized that posterior cruciate ligament reconstructions are often
compromised by associated injuries to the posterolateral structures. Theref
ore, we evaluated a posterior cruciate ligament reconstruction in isolated
and combined injury models using a robotic/ universal force-moment sensor t
esting system. The resulting knee kinematics and the in situ forces in the
native and reconstructed posterior cruciate ligament were determined under
four external loading conditions. In the isolated injury model, reconstruct
ion reduced posterior tibial translation to within 1.5 +/- 1.3 to 2.4 +/- 1
.4 mm of the intact knee at 30 degrees and 90 degrees under a 134-N posteri
or tibial load. In the combined injury model, deficiency of the posterolate
ral structures increased posterior tibial translation of the reconstructed
knee by 6.0 +/- 2.7 mm at 30 degrees and 4.6 +/- 1.5 mm at 90 degrees of fl
exion, External rotation increased up to 14 degrees while varus rotation in
creased up to 7 degrees, In situ forces in the posterior cruciate ligament
graft also increased significantly (by 22% to 150%) for all loading conditi
ons. Our results demonstrate that a graft that restores knee kinematics for
an isolated posterior cruciate ligament deficiency is rendered ineffective
and may be overloaded if the posterolateral structures are deficient, Ther
efore, surgical reconstruction of both structures is recommended in the set
ting of a combined injury.