Case reviews based on autopsy studies have shown that motor vehicle collisi
ons cause between 50 and 90% of traumatic aortic ruptures. Very few studies
have analyzed the nature and severity of the collision forces associated w
ith this injury. Our passenger car study (1984-1991) examined 36 collisions
in which 39 fatally injured victims sustained aortic trauma. In this injur
y group, a disproportionate number of heavy truck and roadside fixed-object
impacts occurred. Vehicle crash forces were generally severe and were eith
er perpendicular or oblique to the vehicle surface. Intrusion into the occu
pant compartment was a significant factor in most of these fatal injuries.
Occupant contact with vehicle interior surfaces was identified in most case
s, and occupant restraints were often ineffective, especially in side colli
sions. The more elderly victims were seen in the least severe collisions.
The most frequent site of aortic rupture was at the isthmus. A majority of
victims had rib/sternal fractures indicating significant chest compression.
Of the various traumatic aortic injury mechanisms proposed in motor vehicl
e impacts, the favored theories in the literature combine features of rapid
deceleration and chest compression. This study supports that predominant i
mpression, concluding that rapid chest deceleration/compression induces tor
sional and shearing forces that result in transverse laceration and rupture
of the aorta, most commonly in the inherently vulnerable isthmus region.