Trauma is a common cause of pulpal damage. In traumatic injuries, the first
priority is to protect the vitality of pulps. But the time between the tra
uma and treatment must be short to preserve vital, noninflamed pulps. The a
im of this study was to investigate the histopathological changes in pulpal
tissues at different time periods after crown fractures. Twenty-three teet
h with enamel and dentin fractures, with and without pulp exposure were eva
luated. The reasons for seeking dental treatment were aesthetic considerati
on, pain, or discomfort The extirpated pulps were histologically prepared f
or microscopical evaluation. There was myelin degeneration surrounding the
axons and edema in the early posttraumatic stages (17 h). In the later stag
es (4 to 20 days), the tissues showed varying degrees of inflammation, and
neuronal degeneration such as intramyelin edema, aberrant myelin synthesis,
and axonal swelling.