Patients with familial adenomatous polyposis (FAP) frequently have extracol
onic manifestations of their disease, Prior reports have indicated an incre
ased prevalence of adrenal lesions in patients with FAP. Although most of t
he adrenal lesions represent nonfunctioning adenomas, some patients have ha
d hypercortisolism due to adrenocortical carcinoma or bilateral nodular hyp
erplasia, We present a case of patient with FAP who had mineralocorticoid e
xcess due to an aldosterone-producing adrenocortical adenoma.