Eleven patients (nine with infarctions and two with primary hematomas) with
isolated thalamic lesions and contralateral asterixis were examined using
a standard electromyographic and neuroimaging protocol. Asterixis was a sho
rt-duration phenomenon associated with a hemiataxia hypesthesia syndrome in
all patients. Electromechanical synchronization was constant for the two s
ilent period types. The anatomic data strongly suggest that ventral lateral
or lateral posterior thalamus are concerned in the pathophysiology of thal
amic asterixis.