The mathematical model described in Part I was solved using " influence lin
e method" combining analytical method and finite element method. Many impor
tant aspects of microcirculatory dynamics were analyzed and discussed. It s
how that interstitial fluid pressure changes its sign twice within one arte
riolar vasomotion period and it is therefore not important that interstitia
l fluid pressure is a little higher or lower than atmospheric pressure; art
eriolar vasomotion can periodically result in lymph formation and interstit
ial total pressure plays an important role in this procedure; local regulat
ion of microcirculation can meet metabolic need some extent in the form of
dynamic equilibrium. The property of arteriole as a " resistant vessel" and
the efficiency of microvascular network as heat exchanger are also shown.
These results shaw that the comprehensive mathematical model developed in P
art I is physiologically reasonable.