Many addiction theories assume that craving plays a central role in the acq
uisition and maintenance of drug dependence. For example, craving is often
depicted as the subjective experience of the motivational state directly re
sponsible for all drinking in the alcoholic. Craving has two prominent feat
ures that must be explained by any viable model of craving. First, craving
tends to be highly situationally specific, readily triggered by stimuli pre
viously associated with drug use. Secondly, craving can persist well beyond
the cessation of drinking in an alcoholic. Conventional theories typically
address craving's cue specificity and persistence by invoking concepts of
classical conditioning. These theories fall into two classes: those that em
phasize withdrawal and those that focus on the positive-incentive propertie
s of drugs. Both types of theories assume that craving processes are repres
ented by the concomitant activation of craving report, drug-seeking and dru
g use, and specific patterns of autonomic responses. However, research fail
s to find more than modest relationships across these putative manifestatio
ns of craving. The cognitive processing model, described in this paper, off
ers a different view of craving's form and function and proposes that drug
use can operate independently of the processes controlling craving. Accordi
ng to this model, addictive drug use is regulated by automatic cognitive pr
ocesses, while craving represents the activation of non-automatic processes
. These non-automatic processes are activated to either aid in completing i
nterrupted drug use or block automatic drug-use sequences. From this perspe
ctive, craving is neither irrelevant nor central to the alcoholic's drug us
e, but rather serves as a cognitive marker of processes that, only in some
instance, may be associated with alcohol seeking and use. The research and
treatment implications of this model's assumptions regarding drug use and c
raving processes are discussed.