A cognitive processing model of alcohol craving and compulsive alcohol use

Citation
St. Tiffany et Ca. Conklin, A cognitive processing model of alcohol craving and compulsive alcohol use, ADDICTION, 95(8), 2000, pp. S145-S153
Citations number
34
Categorie Soggetti
Public Health & Health Care Science","Clinical Psycology & Psychiatry
Journal title
ADDICTION
ISSN journal
09652140 → ACNP
Volume
95
Issue
8
Year of publication
2000
Supplement
2
Pages
S145 - S153
Database
ISI
SICI code
0965-2140(200008)95:8<S145:ACPMOA>2.0.ZU;2-0
Abstract
Many addiction theories assume that craving plays a central role in the acq uisition and maintenance of drug dependence. For example, craving is often depicted as the subjective experience of the motivational state directly re sponsible for all drinking in the alcoholic. Craving has two prominent feat ures that must be explained by any viable model of craving. First, craving tends to be highly situationally specific, readily triggered by stimuli pre viously associated with drug use. Secondly, craving can persist well beyond the cessation of drinking in an alcoholic. Conventional theories typically address craving's cue specificity and persistence by invoking concepts of classical conditioning. These theories fall into two classes: those that em phasize withdrawal and those that focus on the positive-incentive propertie s of drugs. Both types of theories assume that craving processes are repres ented by the concomitant activation of craving report, drug-seeking and dru g use, and specific patterns of autonomic responses. However, research fail s to find more than modest relationships across these putative manifestatio ns of craving. The cognitive processing model, described in this paper, off ers a different view of craving's form and function and proposes that drug use can operate independently of the processes controlling craving. Accordi ng to this model, addictive drug use is regulated by automatic cognitive pr ocesses, while craving represents the activation of non-automatic processes . These non-automatic processes are activated to either aid in completing i nterrupted drug use or block automatic drug-use sequences. From this perspe ctive, craving is neither irrelevant nor central to the alcoholic's drug us e, but rather serves as a cognitive marker of processes that, only in some instance, may be associated with alcohol seeking and use. The research and treatment implications of this model's assumptions regarding drug use and c raving processes are discussed.