Abdominal compartment syndrome is defined by increased intraabdominal press
ure above 20 mmHg with increased pulmonary peak pressure and oliguria. in p
rimary abdominal compartment syndrome the increased intraabdominal pressure
is caused directly by peritonitis, ileus or abdominal and pelvic trauma. S
econdary compartment syndrome is a result of forced closure of the abdomina
l wall after abdominal surgery. The effects are decreased cardiac output, p
ulmonary atelectasis, oliguria to anuria and hepatic as well as intestinal
reduction of perfusion. Effective monitoring is done by standardised measur
ing of urinary bladder pressure. Normal values are between 0 and 7 cm H2O,
after elective laparotomies 5-12 cm H2O. Above 25 cm H2O they are definitel
y pathological. For the prevention and therapy of manifested abdominal comp
artment syndrome the application of a laparostomy using a resorbable mesh i
s recommended. Between 1988 and 1999 we applied a laparostomy to lower the
intraabdominal pressure in 377 patients. In 16% of the cases it was indicat
ed by primary abdominal compartment syndrome with a bladder pressure of 31
+/- 4 cm H2O preoperatively, which could be lowered to 17 +/- 4 cm H2O by l
aparostomy. An early reconstruction of the abdominal wall could be performe
d in 18% of the cases. Conclusions: The abdominal compartment syndrome is a
n often underestimated problem in abdominal surgery involving multiple orga
n systems. The temporary laparostomy lowering intraabdominal pressure rathe
r than a forced closure of the abdominal wall should be used in ail circums
tances.