BACKGROUND: Endurance sports require a variety of physiological adaptations
.
OBJECTIVE: To examine the structural and functional heart adaptations and t
heir hemodynamic implications in triathletes.
PATIENTS AND METHODS: A group of 52 male triathletes was compared with a co
ntrol group of 22 healthy male nonathletes. All of the subjects were given
a bidimensional cardiac Doppler echocardiography examination and administer
ed maximal exercise tests with lactic acid determinations, on a bicycle erg
ometer and a treadmill.
RESULTS: The triathletes showed clear structural and functional heart adapt
ations with concentric and eccentric hypertrophy with evidence of a superno
rmal diastolic left ventricular function. The performance capacity of the t
riathlete differed significantly from that of the control subject. The maxi
mal oxygen consumption and the maximal oxygen consumption per kilogram on t
he bicycle and on the treadmill were significantly higher in the triathlete
s. The same results and conclusions were obtained concerning aerobic capaci
ties and power outputs on a bicycle ergometer at blood lactate concentratio
ns of 2, 3 and 4 mmol/L. The heart rate 6 min alter the start of exercise i
s a significant parameter for the evaluation of the physical condition of a
subject. The lactic acid determinations during the recovery phase enabled
important conclusions to be drawn about the physical condition of the subje
cts.
CONCLUSIONS: The triathletes showed evidence of important structural and fu
nctional heart adaptations with hemodynamic implications. The maximal perfo
rming capacities, on the bicycle as well as on the treadmill, were distinct
ly higher in the triathlete group. Furthermore, the aerobic and anaerobic c
apacities were significantly different between the groups. In this context,
the heart rate 6 min after the start of exercise and the blood lactate con
centrations 20 min alter the maximal exercise test were significant paramet
ers. It was not always the best triathletes who had the most significant st
ructural cardiac adaptations. Thus, the 'athletic heart' syndrome as a phys
iological entity is questioned.