Background: Hyperhomocysteinemia has been increasingly recognized as an imp
ortant risk factor for elevated atherosclerotic vascular disease in chronic
renal failure. We measured in patients with chronic renal failure homocyst
eine and metabolites of its 2 metabolic pathways, transulfuration (cystathi
onine, cysteine) and remethylation (methionine, methylmalonic acid,2-methyl
citric acid).