Viscum modulates a respiratory burst in neutrophils, induced by the chemotactic peptide

The ability of viscum at different concentrations to modulate the respirato ry burst in neutrophils, induced by the chemotactic peptide N-formyl-methio nyl-leucyl-phenylalanine was studied. This does not exclude the possibility that viscum can interact with the receptor of this peptide. The analysis o f the primary structure of viscum revealed elements structurally analogous to the chemotactic peptide. It is assumed that viscum can exhibit the prope rties an antagonist of the receptor of N-formyl-methionyl-leucyl-phenylalan ine, and the mechanism of action of viscum depends on its concentration.