Biofilms form on all hard non-shedding surfaces in a fluid system, i.e. bot
h on teeth and oral implants. As a result of the bacterial challenge, the h
ost responds by mounting a defence mechanism leading to inflammation of the
soft tissues. In the dento-gingival unit, this results in the well-describ
ed lesion of gingivitis. In the implanto-mucosal unit, this inflammation is
termed "mucositis". If plaque is allowed to accumulate for prolonged perio
ds of time, experimental research has demonstrated that "mucositis" may dev
elop into "periimplantitis" affecting the periimplant supporting bone circu
mferentially. Although the bony support may be lost coronally, the implant
still remains osseointegrated and hence, clinically stable. This is the rea
son why mobility represents an insensitive, but specific diagnostic feature
of "periimplantitis". More sensitive and more reliable parameters of devel
oping and existing periimplant infections are "bleeding on probing", "probi
ng depths" and radiographic interpretation of conventional or subtraction r
adiographs. Depending on the diagnosis made continuously during recall visi
ts, a maintenance system termed Cummulative Interceptive Supportive Therapy
(CIST) has been proposed.