Recent research has made striking advances, adding to our understanding of
the aethiopathogenesis of several forms of secondary osteoporosis, includin
g those associated with rheumatic diseases and glucocorticoid treatment. Fu
rthermore in the last 3 years new drugs have been shown to prevent glucocor
ticoid-induced bone loss. Guidelines that are suitable for clinical practic
e have been published. Knowledge of those new opportunities and strategies,
which are briefly reviewed in this article, will reduce the risk of osteop
orotic fractures in rheumatic and non-rheumatic patients.