This study analyzed 15 patients who underwent revision for loosening at the
stem-cement interface. The femoral components were from the same manufactu
rer and had grit-blast roughened surfaces. An apparent radiographic deficie
ncy in the cement mantle was present in at least one zone in 13 patients. I
n 9 of 12 patients with localized osteolysis; the osteolysis developed in a
zone with an apparent radiographic cement mantle defect. Loosening occurre
d due to tension failure of the stem-cement interface followed by axial sub
sidence and movement into relative retroversion. Motion between the stem an
d the cement mantle fueled an abrasive wear mechanism between the roughened
metal surface and the cement mantle, generating excessive metal and cement
particles that gained access to endosteal bone via defects in the cement m
antle and resulting in localized osteolysis.
Although the roughened surface played a central role in these failures, it
is unlikely the layer ofpolymethyl-methacrylate (precoat) played a role in
the mechanism of failure. In some cases, debonding occurred as a result of
tension failure of the metal-precoat interface. In others, tension failure
occurred within the cement mantle, leaving the precoat and some cement from
the mantle on the stems. There was no difference in the mechanism of failu
re of stems with precoat proximally compared to stems with precoat proximal
ly and distally. One stem had no precoat; findings in this patient were ind
istinguishable from the others. The significance of debonding depends on th
e surface roughness of the stem. Debonding carries a poorer prognosis with
a rougher stem surface because of abrasive wear with the generation of nume
rous metal and cement particulates, which lan lead to rapid osteolysis if t
here are cement mantle defects. Stems with a higher metal-cement bond stren
gth may require a higher quality cement mantle for long-term success.