Neonatal camelids can develop hyperglycemia, hypernatremia, and hyperosmola
rity in response to a combination of stress and inadequate water intake.
Clinical signs of this syndrome include a fine head tremor, ataxia, and a b
ase-wide stance of the hind limbs, but biochemical analyses are necessary t
o confirm the diagnosis.
Camelids appear to be susceptible to this syndrome because of a poor insuli
n response to hyperglycemia; hypernatremia results from free water loss ass
ociated with glucose diuresis.
Water loss associated with glucose diuresis may necessitate a higher rate o
f fluid administration in camelids with this syndrome than is typically use
d for treatment of hypernatremia in calves.