Currently the best indicator for surgical treatment of arteriosclerosis is
the degree of stenosis. Although X-ray angiography is currently the standar
d, cost and morbidity are distinct disadvantages. By modelling stenosis and
studying its bio? fluid mechanics, one can apply its results in the field
of arterial disease research. This formed the motivation for this work. A n
on-Newtonian (power law) incompressible Navier-Stokes (NS) solver was devel
oped using the method of operator splitting and artificial compressibility.
The vehicle used is the computational fluid dynamics (CFD) numerical libra
ry FASTFLO. The power-law model developed is then used to do a parametric s
tudy of the effect of 'n' on blood flow mechanics where `n' is the power in
dex that determines the haematocrit of blood. A pulsatile pressure wave ove
r a cardiac cycle of a second was used to simulate transient ? ow over a hy
pothetical two-dimensional stenotic geometry. By comparing the different ve
locity pressure, wall shear stress and viscosity profiles, it has been foun
d when 'n' increases, the vortex formation and peak wall shear stress decre
ases (magnitudes of <1.5 Pa). Since the formation of vortices and low oscil
latory wall shear stress on the stenotic wall is detrimental to the well-be
ing of the arterial tract, it can therefore be inferred that there might be
a relationship between the diseased state of blood (power law) and early g
enesis of atherosclerosis. However, the conclusion of this paper marks the
advent of new research directions in this field of study.