This case involved a 26-month-old boy who had recurrent hemorrhagic venous
infarction caused by venous sinus occlusion. Distension and enlargement of
the cavum septi pellucidi (CSP) and cavum vergae (CV), along with hydroceph
alus, was detected during the course of the disease and was observed to reg
ress together with resolution of the venous occlusion. Venous hypertension
caused by sinus occlusion was thought to be responsible for the disturbed r
esorption of cerebrospinal fluid (CSF) in the CSP and CV in this patient. T
his case is unique because it is the first one to support the hypothesis of
resorption of CSF in the cava by a pressure gradient involving the septal
capillaries and veins.