Heat stress is a major problem in transporting stocker calves with symptoms
of fescue toxicosis. Removing calves from tall fescue pastures and offerin
g diets devoid of endophyte-infected tall fescue could reduce the severity
of toxicosis and precondition calves for transport to the feedlot. In the p
resent experiment, a pasture phase was used to condition yearling steers to
grazing tall fescue and induce symptoms of fescue toxicosis, and a pen pha
se followed to determine effects of implanting at the start of grazing and
protein supplementation (hay only vs hay plus supplement) on shortterm chan
ges in rectal temperature and serum prolactin concentration Neither implant
status nor protein supplementation affected (P > 0.10) white blood cell co
unt or rectal temperature. White blood cell counts at the conclusion of the
pasture phase averaged 8,778 cells/muL and were within a range indicating
no immunological response. Changes in rectal temperature and serum prolacti
n concentration during the pen phase were not influenced (P > 0.10) by impl
anting or supplementation. Initial, rectal temperatures for the pen phase w
ere high (39.9 degreesC) but declined linearly (P < 0.001) over the first 1
06 h and were below a normal temperature (39.2<degrees>C) by 82 h following
removal from tall fescue pastures. Serum prolactin gradually increased (P
< 0.001) to a peak by 82 h and stabilized thereafter. Results indicate that
neither supplemental protein nor an estrogenic implant influenced recovery
indices of fescue toxicosis, whereas removing calves from tall fescue past
ures and excluding dietary tall fescue for 3 to 4 d may alleviate symptoms
of fescue toxicosis.