Athough the myelinated nerve fibers are considered as benign, cases associa
ted with retinal vascular abnormalities (telangiectasis, neovascularization
, vascular occlusions) have recently been reported, suggesting a possible p
athogenic correlation between these elements. Our observation presents a 44
-year-old patient, with a sudden decrease of visual acuity in the right eye
, discal and peridiscal myelinated nerve fibers, associated with cilioretin
al artery occlusion.
The general clinical and laboratory examination; except for a drug stabiliz
ed arterial hypertension, did not reveal other local or general embolic fac
tors. One year later, the decrease in visual acuity had remained unchanged
and the area of the myelinated-nerve fibers had diminished. Based on the as
sociation between the myelinated nerve fibers, the cilioretinal artery occl
usion, the young age of the patient, and the absence of other local or gene
ral embolic factor, we consider there is a possible pathogenic correlation
between these elements. The action of the myelinated nerve fibers can be ex
plained by a mechanical compression, with the disruption of the cilioretina
l artery route, a structurally vulnerable artery.