We report the case of a 28-year-old man with no structural heart disease, w
ho exhibited clearly augmented ST segment elevation in the sight precordial
leads, followed by induction of spontaneous right ventricular outflow trac
t tachycardia with intravenous administration of Class IA antiarrhythmic dr
ugs. The electrophysiologic mechanism of this tachycardia was thought to be
triggered activity due to delayed afterdepolarizations. Due to the existen
ce of substrates that were similar to Brugada syndrome combined with right
ventricular outflow tract tachycardia, this case may represent a subtype of
Brugada syndrome.