Vagal neuropathy after upper respiratory infection: A viral etiology?

Citation
Mr. Amin et Ja. Koufman, Vagal neuropathy after upper respiratory infection: A viral etiology?, AM J OTOLAR, 22(4), 2001, pp. 251-256
Citations number
20
Categorie Soggetti
Otolaryngology
Journal title
AMERICAN JOURNAL OF OTOLARYNGOLOGY
ISSN journal
01960709 → ACNP
Volume
22
Issue
4
Year of publication
2001
Pages
251 - 256
Database
ISI
SICI code
0196-0709(200107/08)22:4<251:VNAURI>2.0.ZU;2-7
Abstract
Purpose: To describe a condition that occurs following an upper respiratory illness, which represents injury to various branches of the vagus nerve. P atients with this condition may present with breathy dysphonia, vocal fatig ue, effortful phonation, odynophonia, cough, globus, and/or dysphagia, last ing long after resolution of the acute viral illness. The patterns of sympt oms and findings in this condition are consistent with the hypothesis that viral infection causes or triggers vagal dysfunction. This so-called postvi ral vagal neuropathy (PVVN) appears to have similarities with other postvir al neuropathic disorders, such as glossopharyngeal neuralgia and Bell's pal sy. Materials and Methods: Five patients were identified with PVVN. Each patien t's chart was reviewed, and elements of the history were recorded. Results: Each of the 5 patients showed different features of PVVN. Conclusions: Respiratory infection can trigger or cause vocal fold paresis, laryngopharyngeal reflux, and neuropathic pain.