Jr. Kim et al., THE RESPONSE TO SUDDEN CHANGE IN VOCAL-TRACT RESISTANCE DURING STOP CONSONANT PRODUCTION, Journal of speech language and hearing research, 40(4), 1997, pp. 848-857
The speech respiratory system is configured in ways that tend to maxim
ize its ability to respond to changes in the airway environment. Intra
oral pressures remain at levels sufficient to generate reliably recogn
ized consonant sounds even in the presence of structural deficits such
as velopharyngeal inadequacy. Similar respiratory compensations occur
when bite blocks and bleed valves are used to vent airway pressures,
The purpose of the present study was to determine the sensitivity of t
he monitoring system psychophysically and to assess its physiological
response to sudden, unanticipated perturbations. Twenty adults were as
ked to produce the utterance /pa/, and a calibrated perturbator valve
permitted air to escape from the oral cavity on randomly selected prod
uctions. Respiratory responses were recorded using PERCI-SARS instrume
ntation. The results indicated that sudden openings of 0.14 cm(2) (SD
= 0.04) were detected by speakers. Compensatory respiratory responses
to suprathreshold pressure-venting occurred rapidly (i.e., 27 ms [SD =
8]) after valve opening. Although peak pressure and area under the pr
essure pulse fell with valve opening, the magnitude of pressure was ne
vertheless sufficient for sound generation. Measurements of the slope
of the rise in intraoral pressure after subthreshold pressure-venting
in 10 participants were compared to measurements obtained From an elas
tic model of the upper airway. The data demonstrated a significant dif
ference between vented and unvented conditions for the model, but not
the participants. This suggests that elastic recoil is actively and un
consciously controlled in humans to compensate For losses in airway pr
essure during speech.