Algodystrophy is characterized initially by an increase in arterial fl
ow, an increase in blood pool, a decrease in microcirculatory velocity
, and a loss of thermoregulatory control. These changes, producing the
typical symptomatology, associated diffuse hand pain, diminished hand
function, edema, and abnormal color and temperature, are believed now
to correspond in most cases to an exaggeration of the normal post-tra
umatic inflammatory responses and are not the consequence of sympathet
ic damage. Algodystrophy may be prevented in most hand surgery patient
s by minimizing the normal postoperative inflammation, by increasing v
enous return (especially by avoiding plaster casts and constrictive dr
essings), by controlling post-traumatic pain, by promoting early activ
e mobilization, and by reassuring the patient. If there is any sign of
algodystrophy, three-phase bone scintigraphy should be obtained and t
reatment instituted immediately.