Authors:
WHITTINGHAM HA
BANTING JD
HEATON JPW
ADAMS MA
Citation: Ha. Whittingham et al., EVIDENCE THAT CGMP AND NOT CAMP-DEPENDENT MECHANISMS REGULATE ENDOTHELIN VASOCONSTRICTOR ACTIVITY IN-VIVO, Journal of hypertension, 16, 1998, pp. 1025-1025
Authors:
WHITTINGHAM HA
BANTING JD
HEATON JPW
ADAMS MA
Citation: Ha. Whittingham et al., ENDOTHELINA B ANTAGONISTS REVERSE THE HYPERSENSITIVITY TO NITRIC-OXIDE DURING NITRIC-OXIDE SYNTHASE BLOCKADE/, Journal of hypertension, 16, 1998, pp. 1236-1236
Authors:
OKABE H
WHITTINGHAM HA
BANTING JD
HEATON JPW
ADAMS MA
Citation: H. Okabe et al., HYPERSENSITIVITY TO VASOCONSTRICTORS FOLLOWING INHIBITION OF NO PRODUCTION IS PREVENTED BY NON-VASODILATOR CONCENTRATIONS OF NITRIC-OXIDE DONORS, The Journal of urology, 159(5), 1998, pp. 363-363
Authors:
ADAMS MA
BANTING JD
MAURICE DH
MORALES A
HEATON JPW
Citation: Ma. Adams et al., VASCULAR CONTROL MECHANISMS IN PENILE ERECTION - PHYLOGENY AND THE INEVITABILITY OF MULTIPLE AND OVERLAPPING SYSTEMS, International journal of impotence research, 9(2), 1997, pp. 85-91
Citation: Jd. Banting et Ma. Adams, ASSESSMENT OF NEURAL, HUMORAL AND LOCAL VASOCONSTRICTOR MECHANISMS INVOLVED IN HYPERTENSION INDUCED BY CHRONIC NITRIC-OXIDE SYNTHASE BLOCKADE, The FASEB journal, 11(3), 1997, pp. 55-55
Citation: Jd. Banting et al., ACUTE HYPERTENSION AFTER NITRIC-OXIDE SYNTHASE INHIBITION IS MEDIATEDPRIMARILY BY INCREASED ENDOTHELIN VASOCONSTRICTION, Journal of hypertension, 14(8), 1996, pp. 975-981
Citation: Jd. Banting et al., HYPERTENSION WITHOUT CARDIAC-HYPERTROPHY DOES NOT INDUCE A CARDIAC BAROREFLEX DEFICIT, Journal of hypertension, 14(10), 1996, pp. 1209-1214
Citation: Jd. Banting et Ma. Adams, INCREASED ENDOTHELIN ACTIVITY PLAYS A PRIMARY ROLE IN HYPERTENSION INDUCED BY ACUTE NITRIC-OXIDE SYNTHASE BLOCKADE, The FASEB journal, 9(3), 1995, pp. 338-338