Citation: Eml. Bastiaanse et al., THE EFFECT OF MEMBRANE CHOLESTEROL CONTENT ON ION-TRANSPORT PROCESSESIN PLASMA-MEMBRANES, Cardiovascular Research, 33(2), 1997, pp. 272-283
Authors:
ATSMA DE
BASTIAANSE EML
VANDERVALK L
VANDERLAARSE A
Citation: De. Atsma et al., LOW EXTERNAL PH LIMITS CELL-DEATH OF ENERGY-DEPLETED CARDIOMYOCYTES BY ATTENUATION OF CA2+ OVERLOAD, American journal of physiology. Heart and circulatory physiology, 39(6), 1996, pp. 2149-2156
Authors:
VERBUNT RJAM
VANDOCKUM WG
BASTIAANSE EML
EGAS JM
VANDERLAARSE A
Citation: Rjam. Verbunt et al., POSTISCHEMIC INJURY IN ISOLATED RAT HEARTS IS NOT AGGRAVATED BY PRIORDEPLETION OF MYOCARDIAL GLUTATHIONE, Molecular and cellular biochemistry, 156(1), 1996, pp. 79-85
Authors:
DEJONGE HW
DEKKERS DHW
BASTIAANSE EML
BEZSTAROSTI K
VANDERLAARSE A
LAMERS JMJ
Citation: Hw. Dejonge et al., EICOSAPENTAENOIC ACID INCORPORATION IN MEMBRANE PHOSPHOLIPIDS MODULATES RECEPTOR-MEDIATED PHOSPHOLIPASE-C AND MEMBRANE FLUIDITY IN RAT VENTRICULAR MYOCYTES IN CULTURE, Journal of Molecular and Cellular Cardiology, 28(5), 1996, pp. 1097-1108
Authors:
VERBUNT RJAM
VANDOCKUM WG
BASTIAANSE EML
EGAS JM
VANDERLAARSE A
Citation: Rjam. Verbunt et al., GLUTATHIONE DISULFIDE AS AN INDEX OF OXIDATIVE STRESS DURING POSTISCHEMIC REPERFUSION IN ISOLATED RAT HEARTS (VOL 144, PG 85, 1995), Molecular and cellular biochemistry, 146(2), 1995, pp. 187-187
Authors:
VERBUNT RJAM
VANDOCKUM WG
BASTIAANSE EML
EGAS JM
VANDERLAARSE A
Citation: Rjam. Verbunt et al., GLUTATHIONE DISULFIDE AS AN INDEX OF OXIDATIVE STRESS DURING POSTISCHEMIC REPERFUSION IN ISOLATED RAT HEARTS, Molecular and cellular biochemistry, 144(1), 1995, pp. 85-93
Authors:
ZHAO SP
BASTIAANSE EML
HAU MF
SMELT AHM
LEUVEN JAG
VANDERLAARSE A
VANTHOOFT FM
Citation: Sp. Zhao et al., SEPARATION OF VLDL-SUBFRACTIONS BY DENSITY GRADIENT ULTRACENTRIFUGATION, The Journal of laboratory and clinical medicine, 125(5), 1995, pp. 641-649
Authors:
ATSMA DE
BASTIAANSE EML
JERZEWSKI A
VANDERVALK LJM
VANDERLAARSE A
Citation: De. Atsma et al., ROLE OF CALCIUM-ACTIVATED NEUTRAL PROTEASE (CALPAIN) IN CELL-DEATH INCULTURED NEONATAL RAT CARDIOMYOCYTES DURING METABOLIC INHIBITION, Circulation research, 76(6), 1995, pp. 1071-1078
Authors:
BASTIAANSE EML
ATSMA DE
VANDERVALK LJM
VANDERLAARSE A
Citation: Eml. Bastiaanse et al., METABOLIC INHIBITION OF CARDIOMYOCYTES CAUSES AN INCREASE IN SARCOLEMMAL FLUIDITY WHICH MAY BE DUE TO LOSS OF CELLULAR CHOLESTEROL, Archives of biochemistry and biophysics, 319(2), 1995, pp. 350-354
Authors:
BASTIAANSE EML
KUIJPERS MC
ATSMA DE
VANDERVALK LJM
VANDERLAARSE A
Citation: Eml. Bastiaanse et al., CELLULAR CHOLESTEROL MODULATION AFFECTS THE INTRACELLULAR CALCIUM-IONCONCENTRATION IN CARDIOMYOCYTES INDEPENDENT OF MEMBRANE FLUIDITY, Journal of the American College of Cardiology, 1994, pp. 10000463-10000463
Authors:
ATSMA DE
BASTIAANSE EML
INCE C
VANDERLAARSE A
Citation: De. Atsma et al., A NOVEL 2-COMPARTMENT CULTURE DISH ALLOWS MICROSCOPIC EVALUATION OF 2DIFFERENT TREATMENTS IN ONE-CELL CULTURE SIMULTANEOUSLY - INFLUENCE OF EXTERNAL PH ON NA+ CA2+ EXCHANGER ACTIVITY IN CULTURED RAT CARDIOMYOCYTES/, Pflugers Archiv, 428(3-4), 1994, pp. 296-299
Authors:
BASTIAANSE EML
VANDERVALKKOKSHOORN LJM
EGASKENNIPHAAS JM
ATSMA DE
VANDERLAARSE A
Citation: Eml. Bastiaanse et al., THE EFFECT OF SARCOLEMMAL CHOLESTEROL CONTENT ON THE TOLERANCE TO ANOXIA IN CARDIOMYOCYTE CULTURES, Journal of Molecular and Cellular Cardiology, 26(5), 1994, pp. 639-648
Authors:
BASTIAANSE EML
ATSMA DE
KUIJPERS MMC
VANDERLAARSE A
Citation: Eml. Bastiaanse et al., SIMVASTATIN-SODIUM DELAYS CELL-DEATH OF ANOXIC CARDIOMYOCYTES BY INHIBITION OF THE NA+ CA2+ EXCHANGER/, FEBS letters, 343(2), 1994, pp. 151-154
Authors:
BASTIAANSE EML
ATSMA DE
KUIJPERS MMC
VANDERLAARSE A
Citation: Eml. Bastiaanse et al., THE EFFECT OF SARCOLEMMAL CHOLESTEROL CONTENT ON INTRACELLULAR CALCIUM-ION CONCENTRATION IN CULTURED CARDIOMYOCYTES, Archives of biochemistry and biophysics, 313(1), 1994, pp. 58-63
Authors:
BASTIAANSE EML
JONGSMA HJ
VANDERLAARSE A
TAKENSKWAK BR
Citation: Eml. Bastiaanse et al., HEPTANOL-INDUCED DECREASE IN CARDIAC GAP JUNCTIONAL CONDUCTANCE IS MEDIATED BY A DECREASE IN THE FLUIDITY OF MEMBRANOUS CHOLESTEROL-RICH DOMAINS, The Journal of membrane biology, 136(2), 1993, pp. 135-145