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Citation: Re. Fleming et al., Mouse strain differences determine severity of iron accumulation in Hfe knockout model of hereditary hemochromatosis, P NAS US, 98(5), 2001, pp. 2707-2711
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Citation: Ca. Sponseller et al., Clinical improvement in patients with decompensated liver disease caused by hepatitis B after treatment with lamivudine, LIVER TRANS, 6(6), 2000, pp. 715-720
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Citation: S. Parkkila et al., Cell surface expression of HFE protein in epithelial cells, macrophages, and monocytes, HAEMATOLOG, 85(4), 2000, pp. 340-345
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Citation: Am. Di Bisceglie et al., Iron reduction as an adjuvant to interferon therapy in patients with chronic hepatitis C who have previously not responded to interferon: A multicenter, prospective, randomized, controlled trial, HEPATOLOGY, 32(1), 2000, pp. 135-138
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Ramm, GA
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Citation: Ga. Ramm et al., Morphology of liver repair following cholestatic liver injury: resolution of ductal hyperplasia, matrix deposition and regression of myofibroblasts, LIVER, 20(5), 2000, pp. 387-396
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Citation: Re. Fleming et al., Transferrin receptor 2: Continued expression in mouse liver in the face ofiron overload and in hereditary hemochromatosis, P NAS US, 97(5), 2000, pp. 2214-2219
Citation: S. Ramrakhiani et Br. Bacon, The new millennium - Advances in viral hepatitis, hepatic disorders, and liver transplantation, MED CLIN NA, 84(5), 2000, pp. 1085
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Authors:
Bassett, SE
Di Bisceglie, AM
Bacon, BR
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Citation: Se. Bassett et al., Effects of iron loading on pathogenicity in hepatitis C virus-infected chimpanzees, HEPATOLOGY, 29(6), 1999, pp. 1884-1892