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Results: 1-16 |
Results: 16
Intestinal responses to xenobiotics
Authors: Carriere, V Chambaz, J Rousset, M
Citation: V. Carriere et al., Intestinal responses to xenobiotics, TOX VITRO, 15(4-5), 2001, pp. 373-378
ApoE protects cortical neurones against neurotoxicity induced by the non-fibrillar C-terminal domain of the amyloid-beta peptide
Authors: Drouet, B Fifre, A Pincon-Raymond, M Vandekerckhove, J Rosseneu, M Gueant, JL Chambaz, J Pillot, T
Citation: B. Drouet et al., ApoE protects cortical neurones against neurotoxicity induced by the non-fibrillar C-terminal domain of the amyloid-beta peptide, J NEUROCHEM, 76(1), 2001, pp. 117-127
Apolipoprotein A-II/A-I ratio is a key determinant in vivo of HDL concentration and formation of pre-beta HDL containing apolipoprotein A-II
Authors: Pastier, D Dugue, S Boisfer, E Atger, V Tran, NQ van Tol, A Chapman, MJ Chambaz, J Laplaud, PM Kalopissis, AD
Citation: D. Pastier et al., Apolipoprotein A-II/A-I ratio is a key determinant in vivo of HDL concentration and formation of pre-beta HDL containing apolipoprotein A-II, BIOCHEM, 40(41), 2001, pp. 12243-12253
Molecular basis of Alzheimer's disease
Authors: Drouet, B Pincon-Raymond, M Chambaz, J Pillot, T
Citation: B. Drouet et al., Molecular basis of Alzheimer's disease, CELL MOL L, 57(5), 2000, pp. 705-715
Transgenic animals with altered high-density lipoprotein composition and functions
Authors: Kalopissis, AD Chambaz, J
Citation: Ad. Kalopissis et J. Chambaz, Transgenic animals with altered high-density lipoprotein composition and functions, CURR OP LIP, 11(2), 2000, pp. 149-153
Cholesterol homeostatic mechanisms in transgenic mice with altered expression of apoproteins A-I, A-II and A-IV
Authors: Kalopissis, AD Chambaz, J
Citation: Ad. Kalopissis et J. Chambaz, Cholesterol homeostatic mechanisms in transgenic mice with altered expression of apoproteins A-I, A-II and A-IV, INT J TISS, 22(2-3), 2000, pp. 67-78
The Socialist Internationale and contemporary concerns
Authors: Chambaz, J
Citation: J. Chambaz, The Socialist Internationale and contemporary concerns, PENSEE, (321), 2000, pp. 115-126
A nonfibrillar form of the fusogenic prion protein fragment [118-135] induces apoptotic cell death in rat cortical neurons
Authors: Pillot, T Drouet, B Pincon-Raymond, RL Vandekerckhove, J Rosseneu, T Chambaz, J
Citation: T. Pillot et al., A nonfibrillar form of the fusogenic prion protein fragment [118-135] induces apoptotic cell death in rat cortical neurons, J NEUROCHEM, 75(6), 2000, pp. 2298-2308
DNA binding specificity and transactivation properties of SREBP-2 bound tomultiple sites on the human apoA-II promoter
Authors: Kan, HY Pissios, P Chambaz, J Zannis, VI
Citation: Hy. Kan et al., DNA binding specificity and transactivation properties of SREBP-2 bound tomultiple sites on the human apoA-II promoter, NUCL ACID R, 27(4), 1999, pp. 1104-1117
The nonfibrillar amyloid beta-peptide induces apoptotic neuronal cell death: Involvement of its C-terminal fusogenic domain
Authors: Pillot, T Drouet, B Queille, S Labeur, C Vandekerckhove, J Rosseneu, M Pincon-Raymond, M Chambaz, J
Citation: T. Pillot et al., The nonfibrillar amyloid beta-peptide induces apoptotic neuronal cell death: Involvement of its C-terminal fusogenic domain, J NEUROCHEM, 73(4), 1999, pp. 1626-1634
Laminin 1 attenuates beta-amyloid peptide A beta(1-40) neurotoxicity of cultured fetal rat cortical neurons
Authors: Drouet, B Pincon-Raymond, M Chambaz, J Pillot, T
Citation: B. Drouet et al., Laminin 1 attenuates beta-amyloid peptide A beta(1-40) neurotoxicity of cultured fetal rat cortical neurons, J NEUROCHEM, 73(2), 1999, pp. 742-749
beta-amyloid peptide interacts specifically with the carboxy-terminal domain of human apolipoprotein E: Relevance to Alzheimer's disease
Authors: Pillot, T Goethals, M Najib, J Labeur, C Lins, L Chambaz, J Brasseur, R Vandekerckhove, J Rosseneu, M
Citation: T. Pillot et al., beta-amyloid peptide interacts specifically with the carboxy-terminal domain of human apolipoprotein E: Relevance to Alzheimer's disease, J NEUROCHEM, 72(1), 1999, pp. 230-237
The -700/-310 fragment of the apolipoprotein A-IV gene combined with the -890/-500 apolipoprotein C-III enhancer is sufficient to direct a pattern ofgene expression similar to that for the endogenous apolipoprotein A-IV gene
Authors: Le Beyec, J Chauffeton, V Kan, HY Janvier, PL Cywiner-Golenzer, C Chatelet, FP Kalopissis, AD Zannis, V Chambaz, J Pincon-Raymond, M Cardot, P
Citation: J. Le Beyec et al., The -700/-310 fragment of the apolipoprotein A-IV gene combined with the -890/-500 apolipoprotein C-III enhancer is sufficient to direct a pattern ofgene expression similar to that for the endogenous apolipoprotein A-IV gene, J BIOL CHEM, 274(8), 1999, pp. 4954-4961
Cooperative binding of upstream stimulatory factor and hepatic nuclear factor 4 drives the transcription of the human apolipoprotein A-II gene
Authors: Ribeiro, A Pastier, D Kardassis, D Chambaz, J Cardot, P
Citation: A. Ribeiro et al., Cooperative binding of upstream stimulatory factor and hepatic nuclear factor 4 drives the transcription of the human apolipoprotein A-II gene, J BIOL CHEM, 274(3), 1999, pp. 1216-1225
Overexpression of human apolipoprotein A-II in mice induces hypertriglyceridemia due to defective very low density lipoprotein hydrolysis
Authors: Boisfer, E Lambert, G Atger, V Tran, NQ Pastier, D Benetollo, C Trottier, JF Beaucamps, I Antonucci, M Laplaud, M Griglio, S Chambaz, J Kalopissis, AD
Citation: E. Boisfer et al., Overexpression of human apolipoprotein A-II in mice induces hypertriglyceridemia due to defective very low density lipoprotein hydrolysis, J BIOL CHEM, 274(17), 1999, pp. 11564-11572
Illegitimate expression of apolipoprotein A-II in Caco-2 cells is due to chromatin organization
Authors: Le Beyec, J Ribeiro, A Schreider, C Chambaz, J Rousset, M Pincon-Raymond, M Cardot, P
Citation: J. Le Beyec et al., Illegitimate expression of apolipoprotein A-II in Caco-2 cells is due to chromatin organization, EXP CELL RE, 247(2), 1999, pp. 373-379
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