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Results:
1-17
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Results: 17
Authors:
Makin, G
Dive, C
Citation: G. Makin et C. Dive, Modulating sensitivity to drug-induced apoptosis: the future for chemotherapy?, BREAST CA R, 3(3), 2001, pp. 150-153
Authors:
Makin, G
Dive, C
Citation: G. Makin et C. Dive, Apoptosis and cancer chemotherapy, TR CELL BIO, 11(11), 2001, pp. S22-S26
Authors:
Griffiths, GJ
Corfe, BM
Savory, P
Leech, S
Esposti, MD
Hickman, JA
Dive, C
Citation: Gj. Griffiths et al., Cellular damage signals promote sequential changes at the N-terminus and BH-1 domain of the pro-apoptotic protein Bak, ONCOGENE, 20(52), 2001, pp. 7668-7676
Authors:
Esposti, MD
Erler, JT
Hickman, JA
Dive, C
Citation: Md. Esposti et al., Bid, a widely expressed proapoptotic protein of the Bcl-2 family, displayslipid transfer activity, MOL CELL B, 21(21), 2001, pp. 7268-7276
Authors:
Makin, GWJ
Corfe, BM
Griffiths, GJ
Thistlethwaite, A
Hickman, JA
Dive, C
Citation: Gwj. Makin et al., Damage-induced Bax N-terminal change, translocation to mitochondria and formation of Bax dimers/complexes occur regardless of cell fate, EMBO J, 20(22), 2001, pp. 6306-6315
Authors:
Khanna, SJ
Brown, R
Whetton, AD
Ball, KL
Dive, C
Citation: Sj. Khanna et al., v-Abl protein-tyrosine kinase up-regulates p21WAF-1 in cell cycle arrestedand proliferating myeloid cells, J BIOL CHEM, 276(14), 2001, pp. 11143-11150
Authors:
Taylor, ST
Hickman, JA
Dive, C
Citation: St. Taylor et al., Epigenetic determinants of resistance to etoposide regulation of Bcl-x(L) and Bax by tumor microenvironmental factors, J NAT CANC, 92(1), 2000, pp. 18-23
Authors:
Corfe, BM
Dive, C
Garrod, DR
Citation: Bm. Corfe et al., Changes in intercellular junctions during apoptosis precede nuclear condensation or phosphatidylserine exposure on the cell surface, CELL DEAT D, 7(2), 2000, pp. 234-235
Authors:
Pierce, A
Spooncer, E
Wooley, S
Dive, C
Francis, JM
Miyan, J
Owen-Lynch, PJ
Dexter, TM
Whetton, AD
Citation: A. Pierce et al., Bcr-Abl protein tyrosine kinase activity induces a loss of p53 protein that mediates a delay in myeloid differentiation, ONCOGENE, 19(48), 2000, pp. 5487-5497
Authors:
Taylor, ST
Hickman, JA
Dive, C
Citation: St. Taylor et al., Survival signals within the tumour microenvironment suppress drug-induced apoptosis: lessons learned from B lymphomas, ENDOCR-R CA, 6(1), 1999, pp. 21-23
Authors:
Benson, RSP
Dive, C
Watson, AJM
Citation: Rsp. Benson et al., Cytoplasmic acidification is not an effector mechanism of VP16 or DEX-induced apoptosis in CEM T leukaemia cells, J CELL SCI, 112(11), 1999, pp. 1755-1760
Authors:
Farrelly, N
Lee, YJ
Oliver, J
Dive, C
Streuli, CH
Citation: N. Farrelly et al., Extracellular matrix regulates apoptosis in mammary epithelium through a control on insulin signaling, J CELL BIOL, 144(6), 1999, pp. 1337-1347
Authors:
Griffiths, GJ
Dubrez, L
Morgan, CP
Jones, NA
Whitehouse, J
Corfe, BM
Dive, C
Hickman, JA
Citation: Gj. Griffiths et al., Cell damage-induced conformational changes of the pro-apoptotic protein bak in vivo precede the onset of apoptosis, J CELL BIOL, 144(5), 1999, pp. 903-914
Authors:
Taylor, ST
Dive, C
Citation: St. Taylor et C. Dive, Drug resistance and the survival niche - Survival signals combine to suppress drug-induced apoptosis in B-lymphoma cells, CANC DRUG, 5, 1999, pp. 205-221
Authors:
Hickman, JA
Dive, C
Citation: Ja. Hickman et C. Dive, Apoptosis and cancer chemotherapy - Preface, CANC DRUG, 5, 1999, pp. IX-X
Authors:
Jones, NA
Dive, C
Citation: Na. Jones et C. Dive, Cell sensitivity assays - Defection of apoptotic cells in vitro using the TUNEL assay, METH MOL M, 28, 1999, pp. 31-38
Authors:
Chen, Q
Takeyama, N
Brady, G
Watson, AJM
Dive, C
Citation: Q. Chen et al., Blood cells with reduced mitochondrial membrane potential and cytosolic cytochrome c can survive and maintain clonogenicity given appropriate signalsto suppress apoptosis, BLOOD, 92(12), 1998, pp. 4545-4553
Risultati:
1-17
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