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Results: 1-11 |
Results: 11
SIGNALING REQUIREMENTS FOR ONCOGENIC FORMS OF THE MET TYROSINE KINASERECEPTOR
Authors: JEFFERS M KOOCHEKPOUR S FISCELLA M SATHYANARAYANA BK VANDEWOUDE GF
Citation: M. Jeffers et al., SIGNALING REQUIREMENTS FOR ONCOGENIC FORMS OF THE MET TYROSINE KINASERECEPTOR, Oncogene, 17(21), 1998, pp. 2691-2700
EVIDENCE FOR A ROLE OF MET-HGF SF DURING RAS-MEDIATED TUMORIGENESIS/METASTASIS/
Authors: WEBB CP TAYLOR GA JEFFERS M FISCELLA M OSKARSSON M RESAU JH WOUDE GFV
Citation: Cp. Webb et al., EVIDENCE FOR A ROLE OF MET-HGF SF DURING RAS-MEDIATED TUMORIGENESIS/METASTASIS/, Oncogene, 17(16), 1998, pp. 2019-2025
THE MUTATIONALLY ACTIVATED MET RECEPTOR MEDIATES MOTILITY AND METASTASIS
Authors: JEFFERS M FISCELLA M WEBB CP ANVER M KOOCHEKPOUR S VANDEWOUDE GF
Citation: M. Jeffers et al., THE MUTATIONALLY ACTIVATED MET RECEPTOR MEDIATES MOTILITY AND METASTASIS, Proceedings of the National Academy of Sciences of the United Statesof America, 95(24), 1998, pp. 14417-14422
CONFORMATION AND SURFACE EXPRESSION OF FREE HLA-CW1 HEAVY-CHAINS IN THE ABSENCE OF BETA(2)-MICROGLOBULIN
Authors: MARTAYAN A FISCELLA M SETINI A CICCARELLI G GAMBARI R FERIOTTO G BERETTA A SICCARDI AG APPELLA E GIACOMINI P
Citation: A. Martayan et al., CONFORMATION AND SURFACE EXPRESSION OF FREE HLA-CW1 HEAVY-CHAINS IN THE ABSENCE OF BETA(2)-MICROGLOBULIN, Human immunology, 53(1), 1997, pp. 23-33
WIP1, A NOVEL HUMAN PROTEIN PHOSPHATASE THAT IS INDUCED IN RESPONSE TO IONIZING-RADIATION IN A P53-DEPENDENT MANNER
Authors: FISCELLA M ZHANG HL FAN SJ SAKAGUCHI K SHEN SF MERCER WE VANDEWOUDE GF OCONNOR PM APPELLA E
Citation: M. Fiscella et al., WIP1, A NOVEL HUMAN PROTEIN PHOSPHATASE THAT IS INDUCED IN RESPONSE TO IONIZING-RADIATION IN A P53-DEPENDENT MANNER, Proceedings of the National Academy of Sciences of the United Statesof America, 94(12), 1997, pp. 6048-6053
A P53-INDEPENDENT PATHWAY FOR ACTIVATION OF WAF1 CIP1 EXPRESSION FOLLOWING OXIDATIVE STRESS/
Authors: RUSSO T ZAMBRANO N ESPOSITO F AMMENDOLA R CIMINO F FISCELLA M JACKMAN J OCONNOR PM ANDERSON CW APPELLA E
Citation: T. Russo et al., A P53-INDEPENDENT PATHWAY FOR ACTIVATION OF WAF1 CIP1 EXPRESSION FOLLOWING OXIDATIVE STRESS/, The Journal of biological chemistry, 270(49), 1995, pp. 29386-29391
THE CARBOXY-TERMINAL SERINE-392 PHOSPHORYLATION SITE OF HUMAN P53 IS NOT REQUIRED FOR WILD-TYPE ACTIVITIES
Authors: FISCELLA M ZAMBRANO N ULLRICH SJ UNGER T LIN D CHO B MERCER WE ANDERSON CW APPELLA E
Citation: M. Fiscella et al., THE CARBOXY-TERMINAL SERINE-392 PHOSPHORYLATION SITE OF HUMAN P53 IS NOT REQUIRED FOR WILD-TYPE ACTIVITIES, Oncogene, 9(11), 1994, pp. 3249-3257
STRUCTURE AND POSTTRANSLATIONAL MODIFICATION OF THE HUMAN P53 PROTEIN
Authors: APPELLA E FISCELLA M ZAMBRANO N ULLRICH SJ SAKAGUCHI K SAKAMOTO H LEWIS MS LIN D MERCER WE ANDERSON CW
Citation: E. Appella et al., STRUCTURE AND POSTTRANSLATIONAL MODIFICATION OF THE HUMAN P53 PROTEIN, Journal of protein chemistry, 13(5), 1994, pp. 499-500
CONSTITUTIVE EXPRESSION OF B-MYB CAN BYPASS P53-INDUCED WAF1 CIP1-MEDIATED G(1) ARREST/
Authors: LIN D FISCELLA M OCONNOR PM JACKMAN J CHEN M LUO LL SALA A TRAVALI S APPELLA E MERCER WE
Citation: D. Lin et al., CONSTITUTIVE EXPRESSION OF B-MYB CAN BYPASS P53-INDUCED WAF1 CIP1-MEDIATED G(1) ARREST/, Proceedings of the National Academy of Sciences of the United Statesof America, 91(21), 1994, pp. 10079-10083
MUTATION OF THE SERINE 15 PHOSPHORYLATION SITE OF HUMAN P53 REDUCES THE ABILITY OF P53 TO INHIBIT CELL-CYCLE PROGRESSION
Authors: FISCELLA M ULLRICH SJ ZAMBRANO N SHIELDS MT LIN D LEESMILLER SP ANDERSON CW MERCER WE APPELLA E
Citation: M. Fiscella et al., MUTATION OF THE SERINE 15 PHOSPHORYLATION SITE OF HUMAN P53 REDUCES THE ABILITY OF P53 TO INHIBIT CELL-CYCLE PROGRESSION, Oncogene, 8(6), 1993, pp. 1519-1528
PHOSPHORYLATION AT SER-15 AND SER-392 IN MUTANT P53-MOLECULES FROM HUMAN TUMORS IS ALTERED COMPARED TO WILD-TYPE P53
Authors: ULLRICH SJ SAKAGUCHI K LEESMILLER SP FISCELLA M MERCER WE ANDERSON CW APPELLA E
Citation: Sj. Ullrich et al., PHOSPHORYLATION AT SER-15 AND SER-392 IN MUTANT P53-MOLECULES FROM HUMAN TUMORS IS ALTERED COMPARED TO WILD-TYPE P53, Proceedings of the National Academy of Sciences of the United Statesof America, 90(13), 1993, pp. 5954-5958
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