Authors:
Weaver, LC
Verghese, P
Bruce, JC
Fehlings, MG
Krenz, NR
Marsh, DR
Citation: Lc. Weaver et al., Autonomic dysreflexia and primary afferent sprouting after clip-compression injury of the rat spinal cord, J NEUROTRAU, 18(10), 2001, pp. 1107-1119
Citation: Mg. Fehlings, Early complications of high-dose methylprednisolone sodium succinate treatment in the follow-up of acute cervical spinal cord injury - Point of view, SPINE, 26(4), 2001, pp. 430-430
Citation: R. Nashmi et Mg. Fehlings, Changes in axonal physiology and morphology after chronic compressive injury of the rat thoracic spinal cord, NEUROSCIENC, 104(1), 2001, pp. 235-251
Citation: S. Casha et al., Oligodendroglial apoptosis occurs along degenerating axons and is associated with Fas and p75 expression following spinal cord injury in the rat, NEUROSCIENC, 103(1), 2001, pp. 203-218
Citation: Mg. Fehlings, Detrimental effects of systemic hyperthermia on locomotor function and histopathological outcome after traumatic spinal cord injury in the rat - Comment, NEUROSURGER, 49(1), 2001, pp. 158-159
Citation: Mg. Fehlings, The effect of groups II and III metabotropic glutamate receptor activationon neuronal injury in a rodent model of traumatic brain injury - Comment, NEUROSURGER, 48(5), 2001, pp. 1126-1126
Citation: Mg. Fehlings, A spinal thecal sac constriction model supports the theory that induced pressure gradients in the cord cause edema and cyst formation - Comment, NEUROSURGER, 48(3), 2001, pp. 645-646
Citation: G. Schwartz et Mg. Fehlings, Evaluation of the neuroprotective effects of sodium channel blockers afterspinal cord injury: improved behavioral and neuroanatomical recovery with riluzole, J NEUROSURG, 94(2), 2001, pp. 245-256
Authors:
Shaker, H
Wang, Y
Loung, D
Balbaa, L
Fehlings, MG
Hassouna, MM
Citation: H. Shaker et al., Role of C-afferent fibres in the mechanism of action of sacral nerve root neuromodulation in chronic spinal cord injury, BJU INT, 85(7), 2000, pp. 905-910
Citation: R. Nashmi et al., Abnormal axonal physiology is associated with altered expression and distribution of Kv1.1 and Kv1.2 K+ channels after chronic spinal cord injury, EUR J NEURO, 12(2), 2000, pp. 491-506
Citation: Sk. Agrawal et al., Role of L- and N-type calcium channels in the pathophysiology of traumaticspinal cord white matter injury, NEUROSCIENC, 99(1), 2000, pp. 179-188
Citation: Mg. Fehlings, Limits of intermittent jugular bulb oxygen saturation monitoring in the management of severe head trauma patients - Comment, NEUROSURGER, 46(5), 2000, pp. 1138-1139
Citation: Mg. Fehlings, The influence of hyperglycemia on neurological outcome in patients with severe head injury - Comment, NEUROSURGER, 46(2), 2000, pp. 343-343
Citation: Pa. Schumacher et al., Pretreatment with calpain inhibitor CEP-4143 inhibits calpain I activationand cytoskeletal degradation, improves neurological function, and enhancesaxonal survival after traumatic spinal cord injury, J NEUROCHEM, 74(4), 2000, pp. 1646-1655
Citation: Sc. Rao et Mg. Fehlings, The optimal radiologic method for assessing spinal canal compromise and cord compression in patients with cervical spinal cord injury - Part I: An evidence-based analysis of the published literature, SPINE, 24(6), 1999, pp. 598-604
Authors:
Fehlings, MG
Rao, SC
Tator, CH
Skaf, G
Arnold, P
Benzel, E
Dickman, C
Cuddy, B
Green, B
Hitchon, P
Northrup, B
Sonntag, V
Wagner, F
Wilberger, J
Citation: Mg. Fehlings et al., The optimal radiologic method for assessing spinal canal compromise and cord compression in patients with cervical spinal cord injury - Part II: Results of a multicenter study, SPINE, 24(6), 1999, pp. 605-613
Citation: Mg. Fehlings, A study of adenosine treatment in experimental acute spinal cord injury - Effect on arachidonic acid metabolites - Point of view, SPINE, 24(2), 1999, pp. 132-132
Citation: Pa. Schumacher et al., Increased calpain I mediated proteolysis, and preferential loss of dephosphorylated NF200, following traumatic spinal cord injury, NEUROSCIENC, 91(2), 1999, pp. 733-744
Citation: Mg. Fehlings, Early treatment with a novel inhibitor of lipid peroxidation (LY341122) improves histopathological outcome after moderate fluid percussion brain injury in rats - Comment, NEUROSURGER, 45(3), 1999, pp. 608-608