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Results: 1-25 | 26-50 | 51-75 | 76-78
Results: 1-25/78
Endogenous nitric oxide mechanisms mediate the stretch dependence of Ca2+ release in cardiomyocytes
Authors: Petroff, MGV Kim, SH Pepe, S Dessy, C Marban, E Balligand, JL Sollott, SJ
Citation: Mgv. Petroff et al., Endogenous nitric oxide mechanisms mediate the stretch dependence of Ca2+ release in cardiomyocytes, NAT CELL BI, 3(10), 2001, pp. 867-873
Maintained contractile reserve in a transgenic mouse model of myocardial stunning
Authors: Kogler, H Soergel, DG Murphy, AM Marban, E
Citation: H. Kogler et al., Maintained contractile reserve in a transgenic mouse model of myocardial stunning, AM J P-HEAR, 280(6), 2001, pp. H2623-H2630
ATP consumption by uncoupled mitochondria activates sarcolemmal K-ATP channels in cardiac myocytes
Authors: Sasaki, N Sato, T Marban, E O'rourke, B
Citation: N. Sasaki et al., ATP consumption by uncoupled mitochondria activates sarcolemmal K-ATP channels in cardiac myocytes, AM J P-HEAR, 280(4), 2001, pp. H1882-H1888
New approaches to antiarrhythmic therapy - Emerging therapeutic applications of the cell biology of cardiac arrhythmias
Authors: Schneider, E Knoll, AG Carmeliet, E Fozzard, HA Hiraoka, M Janse, J Ogawa, S Roden, DM Rosen, MR Rudy, Y Schwartz, PJ Matteo, S Camm, AJ Antzelevitch, C Boyden, PA Catterall, WA Fishman, GI George, AL Izumo, S Jalife, J January, CT Kleber, AG Marban, E Marks, AR Spooner, PM Waldo, AL Weiss, JM Zipes, DP
Citation: E. Schneider et al., New approaches to antiarrhythmic therapy - Emerging therapeutic applications of the cell biology of cardiac arrhythmias, EUR HEART J, 22(23), 2001, pp. 2148-2163
A functional assay for paralytic shellfish toxins that uses recombinant sodium channels
Authors: Velez, P Sierralta, J Alcayaga, C Fonseca, M Loyola, H Johns, DC Tomaselli, GF Marban, E Suarez-Isla, BA
Citation: P. Velez et al., A functional assay for paralytic shellfish toxins that uses recombinant sodium channels, TOXICON, 39(7), 2001, pp. 929-935
Distinct gene-specific mechanisms of arrhythmia revealed by cardiac gene transfer of two long QT disease genes, HERG and KCNE1
Authors: Hoppe, UC Marban, E Johns, DC
Citation: Uc. Hoppe et al., Distinct gene-specific mechanisms of arrhythmia revealed by cardiac gene transfer of two long QT disease genes, HERG and KCNE1, P NAS US, 98(9), 2001, pp. 5335-5340
Pharmacological comparison of native mitochondrial K-ATP channels with molecularly defined surface K-ATP channels
Authors: Liu, YG Ren, GF O'Rourke, B Marban, E Seharaseyon, J
Citation: Yg. Liu et al., Pharmacological comparison of native mitochondrial K-ATP channels with molecularly defined surface K-ATP channels, MOLEC PHARM, 59(2), 2001, pp. 225-230
Functional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytes
Authors: Li, RA Miake, J Hoppe, UC Johns, DC Marban, E Nuss, HB
Citation: Ra. Li et al., Functional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytes, J PHYSL LON, 533(1), 2001, pp. 127-133
Phosphodiesterase inhibitor-mediated potentiation of adenovirus delivery to myocardium
Authors: Nagata, K Marban, E Lawrence, JH Donahue, JK
Citation: K. Nagata et al., Phosphodiesterase inhibitor-mediated potentiation of adenovirus delivery to myocardium, J MOL CEL C, 33(3), 2001, pp. 575-580
Molecular architecture of the volta re-dependent Na channel: Functional evidence for at helices in the pore
Authors: Yamagishi, T Li, RA Hsu, K Marban, E Tomaselli, GF
Citation: T. Yamagishi et al., Molecular architecture of the volta re-dependent Na channel: Functional evidence for at helices in the pore, J GEN PHYSL, 118(2), 2001, pp. 171-181
Kruppel-like factor 4 (Gut-enriched Kruppel-like factor) inhibits cell proliferation by blocking G(1)/S progression of the cell cycle
Authors: Chen, XM Johns, DC Geiman, DE Marban, E Dang, DT Hamlin, G Sun, RG Yang, VW
Citation: Xm. Chen et al., Kruppel-like factor 4 (Gut-enriched Kruppel-like factor) inhibits cell proliferation by blocking G(1)/S progression of the cell cycle, J BIOL CHEM, 276(32), 2001, pp. 30423-30428
Clockwise domain arrangement of the sodium channel revealed by mu-conotoxin (GIIIA) docking orientation
Authors: Li, RA Ennis, IL French, RJ Dudley, SC Tomaselli, GF Marban, E
Citation: Ra. Li et al., Clockwise domain arrangement of the sodium channel revealed by mu-conotoxin (GIIIA) docking orientation, J BIOL CHEM, 276(14), 2001, pp. 11072-11077
Proteomic analysis of pharmacologically preconditioned cardiomyocytes reveals novel phosphorylation of myosin light chain 1
Authors: Arrell, DK Neverova, I Fraser, H Marban, E Van Eyk, JE
Citation: Dk. Arrell et al., Proteomic analysis of pharmacologically preconditioned cardiomyocytes reveals novel phosphorylation of myosin light chain 1, CIRCUL RES, 89(6), 2001, pp. 480-487
Circulation research impact factor sets new record
Authors: Marban, E
Citation: E. Marban, Circulation research impact factor sets new record, CIRCUL RES, 89(2), 2001, pp. 101-101
Sarcoplasmic reticulum Ca2+ ATPase (SERCA) 1a structurally substitutes forSERCA2a in the cardiac sarcoplasmic reticulum and increases cardiac Ca2+ handling capacity
Authors: Lalli, MJ Yong, J Prasad, V Hashimoto, K Plank, D Babu, GJ Kirkpatrick, D Walsh, RA Sussman, M Yatani, A Marban, E Periasamy, M
Citation: Mj. Lalli et al., Sarcoplasmic reticulum Ca2+ ATPase (SERCA) 1a structurally substitutes forSERCA2a in the cardiac sarcoplasmic reticulum and increases cardiac Ca2+ handling capacity, CIRCUL RES, 89(2), 2001, pp. 160-167
Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca2+ overload during simulated ischemia and reperfusion - Possible mechanism of cardioprotection
Authors: Murata, M Akao, M O'Rourke, B Marban, E
Citation: M. Murata et al., Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca2+ overload during simulated ischemia and reperfusion - Possible mechanism of cardioprotection, CIRCUL RES, 89(10), 2001, pp. 891-898
Molecular interactions between two long-QT syndrome gene products, HERG and KCNE2, rationalized by in vitro and in silico analysis
Authors: Mazhari, R Greenstein, JL Winslow, RL Marban, E Nuss, HB
Citation: R. Mazhari et al., Molecular interactions between two long-QT syndrome gene products, HERG and KCNE2, rationalized by in vitro and in silico analysis, CIRCUL RES, 89(1), 2001, pp. 33-38
Functional roles of cardiac and vascular ATP-sensitive potassium channels clarified by Kir6.2-knockout mice
Authors: Suzuki, M Li, RA Miki, T Uemura, H Sakamoto, N Ohmoto-Sekine, Y Tamagawa, M Ogura, T Seino, S Marban, E Nakaya, H
Citation: M. Suzuki et al., Functional roles of cardiac and vascular ATP-sensitive potassium channels clarified by Kir6.2-knockout mice, CIRCUL RES, 88(6), 2001, pp. 570-577
Mitochondrial ATP-sensitive potassium channels inhibit apoptosis induced by oxidative stress in cardiac cells
Authors: Akao, M Ohler, A O'Rourke, B Marban, E
Citation: M. Akao et al., Mitochondrial ATP-sensitive potassium channels inhibit apoptosis induced by oxidative stress in cardiac cells, CIRCUL RES, 88(12), 2001, pp. 1267-1275
New approaches to antiarrhythmic therapy, part I - Emerging therapeutic applications of the cell biology of cardiac arrhythmias
Authors: Carmeliet, E Fozzard, HA Hiraoka, M Janse, MJ Ogawa, S Roden, DM Rosen, MR Rudy, Y Schwartz, PJ Matteo, PS Antzelevitch, C Boyden, PA Catterall, WA Fishman, GI George, AL Izumo, S Jalife, J January, CT Kleber, AG Marban, E Marks, AR Spooner, PM Waldo, AL Weiss, JM Zipes, DLP
Citation: E. Carmeliet et al., New approaches to antiarrhythmic therapy, part I - Emerging therapeutic applications of the cell biology of cardiac arrhythmias, CIRCULATION, 104(23), 2001, pp. 2865-2873
Allopurinol improves myocardial efficiency in patients with idiopathic dilated cardiomyopathy
Authors: Cappola, TP Kass, DA Nelson, GS Berger, RD Rosas, GO Kobeissi, ZA Marban, E Hare, JM
Citation: Tp. Cappola et al., Allopurinol improves myocardial efficiency in patients with idiopathic dilated cardiomyopathy, CIRCULATION, 104(20), 2001, pp. 2407-2411
Sudden cardiac death, genes, and arrhythmogenesis - Consideration of new population and mechanistic approaches from a National Heart, Lung, and BloodInstitute Workshop, part II
Authors: Spooner, PM Albert, C Benjamin, EJ Boineau, R Elston, RC George, AL Jouven, X Kuller, LH MacCluer, JW Marban, E Muller, JE Schwartz, PJ Siscovick, DS Tracy, RP Zareba, W Zipes, DP
Citation: Pm. Spooner et al., Sudden cardiac death, genes, and arrhythmogenesis - Consideration of new population and mechanistic approaches from a National Heart, Lung, and BloodInstitute Workshop, part II, CIRCULATION, 103(20), 2001, pp. 2447-2452
Sudden cardiac death, genes, and arrhythmogenesis - Consideration of new population and mechanistic approaches from a national heart, lung, and bloodinstitute workshop, part I
Authors: Spooner, PM Albert, C Benjamin, EJ Boineau, R Elston, RC George, AL Jouven, X Kuller, LH MacCluer, JW Marban, E Muller, JE Schwartz, PJ Siscovick, DS Tracy, RP Zareba, W Zipes, DP
Citation: Pm. Spooner et al., Sudden cardiac death, genes, and arrhythmogenesis - Consideration of new population and mechanistic approaches from a national heart, lung, and bloodinstitute workshop, part I, CIRCULATION, 103(19), 2001, pp. 2361-2364
Latent specificity of molecular recognition in sodium channels engineered to discriminate between two "indistinguishable'' mu-conotoxins
Authors: Li, RA Ennis, IL Tomaselli, GF French, RJ Marban, E
Citation: Ra. Li et al., Latent specificity of molecular recognition in sodium channels engineered to discriminate between two "indistinguishable'' mu-conotoxins, BIOCHEM, 40(20), 2001, pp. 6002-6008
Pharmacologically induced preconditioning with diazoxide: A novel approachto brain protection
Authors: Shake, JG Peck, EA Marban, E Gott, VL Johnston, MV Troncoso, JC Redmond, JM Baumgartner, WA
Citation: Jg. Shake et al., Pharmacologically induced preconditioning with diazoxide: A novel approachto brain protection, ANN THORAC, 72(6), 2001, pp. 1849-1854
Risultati: 1-25 | 26-50 | 51-75 | 76-78