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He, H
Hirokawa, Y
Manser, E
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Authors:
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Catimel, B
Nice, E
Maruta, H
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Treutlein, HR
Citation: J. Zeng et al., Design of inhibitors of Ras-Raf interaction using a computational combinatorial algorithm, PROTEIN ENG, 14(1), 2001, pp. 39-45
Authors:
He, H
Levitzki, A
Zhu, HJ
Walker, F
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Citation: H. He et al., Platelet-derived growth factor requires epidermal growth factor receptor to activate p21-activated kinase family kinases, J BIOL CHEM, 276(29), 2001, pp. 26741-26744
Authors:
Sachdev, P
Jiang, YX
Li, WQ
Miki, T
Maruta, H
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Wang, LH
Citation: P. Sachdev et al., Differential requirement for Rho family GTPases in an oncogenic insulin-like growth factor-I receptor-induced cell transformation, J BIOL CHEM, 276(28), 2001, pp. 26461-26471
Citation: H. He et al., An anti-Ras cancer potential of PP1, an inhibitor specific for Src family kinases: In vitro and in vivo studies, CANCER J, 6(4), 2000, pp. 243-248
Authors:
Tikoo, A
Czekay, S
Viars, C
White, S
Heath, JK
Arden, K
Maruta, H
Citation: A. Tikoo et al., P190-A, a human tumor suppressor gene, maps to the chromosomal region 19q13.3 that is reportedly deleted in some gliomas, GENE, 257(1), 2000, pp. 23-31
Authors:
Wadhwa, R
Sugihara, T
Yoshida, A
Nomura, H
Reddel, RR
Simpson, R
Maruta, H
Kaul, SC
Citation: R. Wadhwa et al., Selective toxicity of MKT-077 to cancer cells is mediated by its binding to the hsp70 family protein mot-2 and reactivation of p53 function, CANCER RES, 60(24), 2000, pp. 6818-6821
Authors:
Tikoo, A
Cutler, H
Lo, SH
Chen, LB
Maruta, H
Citation: A. Tikoo et al., Treatment of Ras-induced cancers by the F-actin cappers tensin and chaetoglobosin K, in combination with the caspase-1 inhibitor N1445, CA J SCI AM, 5(5), 1999, pp. 293-300
Authors:
Zeng, J
Fridman, M
Maruta, H
Treutlein, HR
Simonson, T
Citation: J. Zeng et al., Protein-protein recognition: An experimental and computational study of the R89K mutation in Raf and its effect on Ras binding, PROTEIN SCI, 8(1), 1999, pp. 50-64
Authors:
Nur-E-Kamal, MSA
Kamal, JM
Qureshi, MM
Maruta, H
Citation: Msa. Nur-e-kamal et al., The CDC42-specific inhibitor derived from ACK-1 blocks v-Ha-Ras-induced transformation, ONCOGENE, 18(54), 1999, pp. 7787-7793