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Citation: B. Fisher et al., LUMPECTOMY AND RADIATION-THERAPY FOR THE TREATMENT OF INTRADUCTAL BREAST-CANCER - FINDINGS FROM NATIONAL SURGICAL ADJUVANT BREAST AND BOWELPROJECT B-17, Journal of clinical oncology, 16(2), 1998, pp. 441-452
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Citation: S. Gambaryan et al., ENDOGENOUS OR OVEREXPRESSED CGMP-DEPENDENT PROTEIN-KINASES INHIBIT CAMP-DEPENDENT RENIN RELEASE FROM RAT ISOLATED-PERFUSED KIDNEY, MICRODISSECTED GLOMERULI, AND ISOLATED JUXTAGLOMERULAR CELLS, Proceedings of the National Academy of Sciences of the United Statesof America, 95(15), 1998, pp. 9003-9008
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Citation: C. Schmitt et al., INVESTIGATION OF FACTOR VIII-ANTIGEN LEVELS OF PATIENTS WITH HEMOPHILIA-A BY A NEW HIGHLY SENSITIVE ENZYME-LINKED-IMMUNOSORBENT-ASSAY (ELISA), Thrombosis and haemostasis, 1997, pp. 120-120
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Citation: A. Haack et al., IN-VITRO EXPRESSION STUDIES OF A NEW FUNCTIONALLY ACTIVE B-DOMAIN-TRUNCATED RECOMBINANT FVIII PROTEIN, Thrombosis and haemostasis, 1997, pp. 931-931
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Citation: W. Poller et al., STABILIZATION OF TRANSGENE EXPRESSION BY INCORPORATION OF E3 REGION GENES INTO AN ADENOVIRAL FACTOR-IX VECTOR AND BY TRANSIENT ANTI-CD4 TREATMENT OF THE HOST, Gene therapy, 3(6), 1996, pp. 521-530
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Citation: M. Paul et al., THE ROLE OF ENDOTHELINS AND THEIR RECEPTORS IN THE CARDIOVASCULAR-SYSTEM, Naunyn-Schmiedeberg's archives of pharmacology, 354(4), 1996, pp. 5-5
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Citation: Vn. Kalinin et al., A POINT MUTATION IN THE ND1 MITOCHONDRIAL GENE IDENTIFIED IN A TYPE-II DIABETIC PATIENT, Genetika, 31(8), 1995, pp. 1180-1182
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Citation: J. Hager et al., A MISSENSE MUTATION IN THE GLUCAGON RECEPTOR GENE IS LINKED TO FAMILIAL NIDDM, Diabetologia, 37, 1994, pp. 10000016-10000016
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Citation: J. Hager et al., 6 MUTATIONS IN THE GLUCOKINASE GENE IDENTIFIED IN MODY BY USING A NONRADIOACTIVE SENSITIVE SCREENING TECHNIQUE, Diabetes, 43(5), 1994, pp. 730-733
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Citation: Jp. Faber et al., IDENTIFICATION AND DNA-SEQUENCE ANALYSIS OF 15 NEW ALPHA(1)-ANTITRYPSIN VARIANTS, INCLUDING 2 PI-ASTERISK-Q0 ALLELES AND ONE DEFICIENT PI-ASTERISK-M ALLELE, American journal of human genetics, 55(6), 1994, pp. 1113-1121
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Citation: W. Poller et al., A LEUCINE-TO-PROLINE SUBSTITUTION CAUSES A DEFECTIVE ALPHA(1)-ANTICHYMOTRYPSIN ALLELE ASSOCIATED WITH FAMILIAL OBSTRUCTIVE LUNG-DISEASE, Genomics, 17(3), 1993, pp. 740-743
Citation: W. Poller et H. Schatz, MOLECULAR-GENETIC ANALYSIS OF NIDDM - STATUS AND PROSPECTS, Experimental and clinical endocrinology, 101(2), 1993, pp. 58-68
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Citation: Jp. Faber et al., THE MOLECULAR-BASIS OF ALPHA(1)-ANTICHYMOTRYPSIN DEFICIENCY IN A HETEROZYGOTE WITH LIVER AND LUNG-DISEASE, Journal of hepatology, 18(3), 1993, pp. 313-321
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Citation: B. Fisher et al., LUMPECTOMY COMPARED WITH LUMPECTOMY AND RADIATION-THERAPY FOR THE TREATMENT OF INTRADUCTAL BREAST-CANCER, The New England journal of medicine, 328(22), 1993, pp. 1581-1586
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Citation: J. Hager et al., GENE SCANNING OF GLUCOKINASE IN GERMAN EARLY AND LATE AGE-OF-ONSET NIDDM FAMILIES, Diabetologia, 36, 1993, pp. 10000084-10000084
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Citation: W. Poller et al., MOLECULAR-BASIS OF A DEFECTIVE ALPHA-1-ANTICHYMOTRYPSIN PHENOTYPE (BOCHUM-I) ASSOCIATED WITH FAMILIAL CHRONIC OBSTRUCTIVE LUNG-DISEASE, The American review of respiratory disease, 147(4), 1993, pp. 677-677
Authors:
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POLLER W
WIETHEGE T
BOHM S
BRAUN DE
BAUR X
Citation: B. Voss et al., ELEVATED LAMINI P1 CONCENTRATIONS IN SERA OF PATIENTS WITH CHRONIC OBSTRUCTIVE PULMONARY-DISEASE, The American review of respiratory disease, 147(4), 1993, pp. 869-869