AAAAAA

   
Results: 1-10 |
Results: 10
The murine type 1 diabetes loci, Idd1, Idd3, Idd5, Idd9, and idd17/10/18, do not control thymic CD4(-)CD8(-)/TCR alpha beta(+) deficiency in the nonobese diabetic mouse
Authors: Lord, CJ Howlett, S Lyons, PA Peterson, LB Wicker, LS Todd, JA
Citation: Cj. Lord et al., The murine type 1 diabetes loci, Idd1, Idd3, Idd5, Idd9, and idd17/10/18, do not control thymic CD4(-)CD8(-)/TCR alpha beta(+) deficiency in the nonobese diabetic mouse, MAMM GENOME, 12(2), 2001, pp. 175-176
Statistical modeling of interlocus interactions in a complex disease: Rejection of the multiplicative model of epistasis in type 1 diabetes
Authors: Cordell, HJ Todd, JA Hill, NJ Lord, CJ Lyons, PA Peterson, LB Wicker, LS Clayton, DG
Citation: Hj. Cordell et al., Statistical modeling of interlocus interactions in a complex disease: Rejection of the multiplicative model of epistasis in type 1 diabetes, GENETICS, 158(1), 2001, pp. 357-367
Mapping by genetic interaction - High-resolution congenic mapping of the type 1 diabetes loci Idd10 and Idd18 in the NOD mouse
Authors: Lyons, PA Armitage, N Lord, CJ Phillips, MS Todd, JA Peterson, LB Wicker, LS
Citation: Pa. Lyons et al., Mapping by genetic interaction - High-resolution congenic mapping of the type 1 diabetes loci Idd10 and Idd18 in the NOD mouse, DIABETES, 50(11), 2001, pp. 2633-2637
Congenic mapping of the type 1 diabetes locus, ldd3, to a 780-kb region ofmouse chromosome 3: Identification of a candidate segment of ancestral DNAby haplotype mapping
Authors: Lyons, PA Armitage, N Argentina, F Denny, P Hill, NJ Lord, CJ Wilusz, MB Peterson, LB Wicker, LS Todd, JA
Citation: Pa. Lyons et al., Congenic mapping of the type 1 diabetes locus, ldd3, to a 780-kb region ofmouse chromosome 3: Identification of a candidate segment of ancestral DNAby haplotype mapping, GENOME RES, 10(4), 2000, pp. 446-453
The NOD Idd9 genetic interval influences the pathogenicity of insulitis and contains molecular variants of Cd30, Tnfr2, and Cd137
Authors: Lyons, PA Hancock, WW Denny, P Lord, CJ Hill, NJ Armitage, N Siegmund, T Todd, JA Phillips, MS Hess, JF Chen, SL Fischer, PA Peterson, LB Wicker, LS
Citation: Pa. Lyons et al., The NOD Idd9 genetic interval influences the pathogenicity of insulitis and contains molecular variants of Cd30, Tnfr2, and Cd137, IMMUNITY, 13(1), 2000, pp. 107-115
Differential glycosylation of interleukin 2, the molecular basis for the NOD Idd3 type 1 diabetes gene?
Authors: Podolin, PL Wilusz, MB Cubbon, RM Pajvani, U Lord, CJ Todd, JA Peterson, LB Wicker, LS Lyons, PA
Citation: Pl. Podolin et al., Differential glycosylation of interleukin 2, the molecular basis for the NOD Idd3 type 1 diabetes gene?, CYTOKINE, 12(5), 2000, pp. 477-482
Analysis of the mouse CD30 gene - A candidate for the NOD mouse type 1 diabetes locus Idd9.2
Authors: Siegmund, T Armitage, N Wicker, LS Peterson, LB Todd, JA Lyons, PA
Citation: T. Siegmund et al., Analysis of the mouse CD30 gene - A candidate for the NOD mouse type 1 diabetes locus Idd9.2, DIABETES, 49(9), 2000, pp. 1612-1616
NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humans
Authors: Hill, NJ Lyons, PA Armitage, N Todd, JA Wicker, LS Peterson, LB
Citation: Nj. Hill et al., NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humans, DIABETES, 49(10), 2000, pp. 1744-1747
QTL influencing autoimmune diabetes and encephalomyelitis map to a 0.15-cMregion containing I/2
Authors: Encinas, JA Wicker, LS Peterson, LB Mukasa, A Teuscher, C Sobel, R Weiner, HL Seidman, CE Seidman, JG Kuchroo, VK
Citation: Ja. Encinas et al., QTL influencing autoimmune diabetes and encephalomyelitis map to a 0.15-cMregion containing I/2, NAT GENET, 21(2), 1999, pp. 158-160
Potent immunosuppressive C32-O-arylethyl ether derivatives of ascomycin with reduced toxicity.
Authors: Armstrong, HM Wong, F Holmes, MA Sinclair, PJ Goulet, MT Dumont, FJ Staruch, M Koprak, S Peterson, LB Rosa, R Wilusz, MB Wiederrecht, GJ Cryan, JG Wyvratt, MJ Parsons, WH
Citation: Hm. Armstrong et al., Potent immunosuppressive C32-O-arylethyl ether derivatives of ascomycin with reduced toxicity., BIOORG MED, 9(14), 1999, pp. 2089-2094
Risultati: 1-10 |