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Citation: T. Lapvetelainen et al., More knee joint osteoarthritis (OA) in mice after inactivation of one allele of type II procollagen gene but less OA after lifelong voluntary wheel running exercise, OSTEO CART, 9(2), 2001, pp. 152-160
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Citation: Sw. Li et al., Targeted disruption of Col11a2 produces a mild cartilage phenotype in transgenic mice: Comparison with the human disorder otospondylomegaepiphyseal dysplasia (OSMED), DEV DYNAM, 222(2), 2001, pp. 141-152
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Citation: Ej. Schwarz et al., Rat marrow stromal cells rapidly transduced with a self-inactivating retrovirus synthesize L-DOPA in vitro, GENE THER, 8(16), 2001, pp. 1214-1223
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Citation: Sw. Li et al., Transgenic mice with inactive alleles for procollagen N-proteinase (ADAMTS-2) develop fragile skin and male sterility, BIOCHEM J, 355, 2001, pp. 271-278
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Citation: Em. Horwitz et al., Clinical responses to bone marrow transplantation in children with severe osteogenesis imperfecta, BLOOD, 97(5), 2001, pp. 1227-1231
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Citation: Ww. Deng et al., In vitro differentiation of human marrow stromal cells into early progenitors of neural cells by conditions that increase intracellular cyclic AMP, BIOC BIOP R, 282(1), 2001, pp. 148-152
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Citation: Mm. Hyttinen et al., Inactivation of one allele of the type II collagen gene alters the collagen network in murine articular cartilage and makes cartilage softer, ANN RHEUM D, 60(3), 2001, pp. 262-268
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Citation: Jt. Vuoristo et al., Sequence and genomic organization of the human G-protein Golf alpha gene (GNAL) on chromosome 18p11, a susceptibility region for bipolar disorder andschizophrenia, MOL PSYCHI, 5(5), 2000, pp. 495-501
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Citation: Ia. Kramerova et al., Papilin in development; a pericellular protein with a homology to the ADAMTS metalloproteinases, DEVELOPMENT, 127(24), 2000, pp. 5475-5485
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Citation: Dj. Prockop et al., Potential use of stem cells from bone marrow to repair the extracellular matrix and the central nervous system, BIOCH SOC T, 28, 2000, pp. 341-345
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Citation: Cm. Yuan et al., Lack of a phenotype in transgenic mice aberrantly expressing COL2A1 mRNA because of highly selective post-transcriptional down-regulation, BIOCHEM J, 345, 2000, pp. 377-384
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Citation: J. Korkko et al., Widely distributed mutations in the COL2A1 gene produce achondrogenesis type II/hypochondrogenesis, AM J MED G, 92(2), 2000, pp. 95-100
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Citation: Dc. Colter et al., Rapid expansion of recycling stem cells in cultures of plastic-adherent cells from human bone marrow, P NAS US, 97(7), 2000, pp. 3213-3218
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Citation: Em. Horwitz et al., Transplantability and therapeutic effects of bone marrow-derived mesenchymal cells in children with osteogenesis imperfecta, NAT MED, 5(3), 1999, pp. 309-313
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Citation: Wt. Mcguirt et al., Mutations in COL11A2 cause non-syndromic hearing loss (DFNA13), NAT GENET, 23(4), 1999, pp. 413-419
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Citation: Ej. Schwarz et al., Multipotential marrow stromal cells transduced to produce L-DOPA: Engraftment in a rat model of Parkinson disease, HUM GENE TH, 10(15), 1999, pp. 2539-2549
Authors:
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Prockop, DJ
Fertala, A
Citation: E. Adachi et al., Collagen II containing a Cys substitution for Arg-alpha 1-519. Analysis byatomic force microscopy demonstrates that mutated monomers alter the topography of the surface of collagen II fibrils, MATRIX BIOL, 18(2), 1999, pp. 189-196
Citation: Dj. Prockop, Genetic trail of osteoporosis: Candidate genes versus genome screens? Small families versus large families?, J BONE MIN, 14(12), 1999, pp. 2000-2001