Citation: Ke. Berge et K. Berg, POLYMORPHISMS AT THE ANGIOTENSINOGEN (AGT) AND ANGIOTENSIN-II TYPE-1 RECEPTOR (AT1R) LOCI AND NORMAL BLOOD-PRESSURE, Clinical genetics, 53(3), 1998, pp. 214-219
Citation: Ke. Berge et K. Berg, CARDIOVASCULAR RISK-FACTORS IN PEOPLE WITH DIFFERENT GENOTYPES IN THEINSERTION DELETION (I/D) POLYMORPHISM AT THE LOCUS FOR ANGIOTENSIN-I-CONVERTING-ENZYME (ACE)/, Clinical genetics, 52(6), 1997, pp. 422-426
Authors:
BERGE KE
DJUROVIC S
MULLER HJ
ALESTROM P
BERG K
Citation: Ke. Berge et al., STUDIES ON EFFECTS OF LP(A) LIPOPROTEIN ON GENE-EXPRESSION IN ENDOTHELIAL-CELLS IN-VITRO, Clinical genetics, 52(5), 1997, pp. 314-325
Authors:
BERGE KE
BAKKEN A
BOHN M
ERIKSSEN J
BERG K
Citation: Ke. Berge et al., A DNA POLYMORPHISM AT THE ANGIOTENSIN-II TYPE-1 RECEPTOR (ATIR) LOCUSAND MYOCARDIAL-INFARCTION, Clinical genetics, 52(2), 1997, pp. 71-76
Authors:
BERGE KE
BAKKEN A
BOHN M
ERIKSSEN J
BERG K
Citation: Ke. Berge et al., ANALYSES OF MUTATIONS IN THE HUMAN RENAL KALLIKREIN (HKLK1) GENE AND THEIR POSSIBLE RELEVANCE TO BLOOD-PRESSURE REGULATION AND RISK OF MYOCARDIAL-INFARCTION, Clinical genetics, 52(2), 1997, pp. 86-95
Citation: V. Berge et al., VITRONECTIN MODULATES THE EXPRESSION OF COMPLEMENT COMPONENTS OF THE TERMINAL PATHWAY SYNTHESIZED BY HUMAN UMBILICAL VEIN ENDOTHELIAL-CELLSIN-VITRO, APMIS. Acta pathologica, microbiologica et immunologica Scandinavica, 104(7-8), 1996, pp. 523-530
Citation: V. Berge et al., INTERLUEKIN-1-ALPHA, INTERLEUKIN-6 AND TUMOR-NECROSIS-FACTOR-ALPHA INCREASE THE SYNTHESIS AND EXPRESSION OF THE FUNCTIONAL ALTERNATIVE AND TERMINAL COMPLEMENT PATHWAYS BY HUMAN UMBILICAL VEIN ENDOTHELIAL-CELLSIN-VITRO, APMIS. Acta pathologica, microbiologica et immunologica Scandinavica, 104(3), 1996, pp. 213-219
Citation: Ke. Berge et K. Berg, NO EFFECT ON BLOOD-PRESSURE LEVEL OR VARIABILITY OF POLYMORPHISMS IN DNA AT THE LOCUS FOR ATRIAL-NATRIURETIC-FACTOR (ANF), Clinical genetics, 46(6), 1994, pp. 433-435
Citation: Ke. Berge et K. Berg, NO EFFECT OF A BGLI POLYMORPHISM AT THE RENIN (REN) LOCUS ON BLOOD-PRESSURE LEVEL OR VARIABILITY, Clinical genetics, 46(6), 1994, pp. 436-438
Citation: Ke. Berge et al., DNA POLYMORPHISM AT THE LOCUS FOR ANGIOTENSINOGEN-I-CONVERTING ENZYMEIN NORWEGIAN PATIENTS WITH MYOCARDIAL-INFARCTION AND CONTROLS, Clinical genetics, 46(1), 1994, pp. 102-104
Citation: Ke. Berge et K. Berg, NO EFFECT OF INSERTION DELETION POLYMORPHISM AT THE ACE LOCUS ON NORMAL BLOOD-PRESSURE LEVEL OR VARIABILITY, Clinical genetics, 45(4), 1994, pp. 169-174
Authors:
BOHN M
BERGE KE
BAKKEN A
ERIKSSEN J
BERG K
Citation: M. Bohn et al., INSERTION DELETION (I D) POLYMORPHISM AT THE LOCUS FOR ANGIOTENSIN-I-CONVERTING ENZYME AND MYOCARDIAL-INFARCTION/, Clinical genetics, 44(6), 1993, pp. 292-297
Authors:
BOHN M
BERGE KE
BAKKEN A
ERIKSSEN J
BERG K
Citation: M. Bohn et al., INSERTION DELETION (I D) POLYMORPHISM AT THE LOCUS FOR ANGIOTENSIN I-CONVERTING ENZYME AND PARENTAL HISTORY OF MYOCARDIAL-INFARCTION/, Clinical genetics, 44(6), 1993, pp. 298-301
Citation: Ke. Berge et K. Berg, NO EFFECT OF TAQI POLYMORPHISM AT THE HUMAN RENAL KALLIKREIN (KLK1) LOCUS ON NORMAL BLOOD-PRESSURE LEVEL OR VARIABILITY, Clinical genetics, 44(4), 1993, pp. 196-202