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Results: 1-19 |
Results: 19
Constitutive Cdc25B tyrosine phosphatase activity in adult brain neurons with M phase-type alterations in Alzheimer's disease
Authors: Vincent, I Bu, B Hudson, K Husseman, J Nochlin, D Jin, LW
Citation: I. Vincent et al., Constitutive Cdc25B tyrosine phosphatase activity in adult brain neurons with M phase-type alterations in Alzheimer's disease, NEUROSCIENC, 105(3), 2001, pp. 639-650
Hyperphosphorylation of RNA polymerase II and reduced neuronal RNA levels precede neurofibrillary tangles in Alzheimer disease
Authors: Husseman, JW Hallows, JL Bregman, DB Leverenz, JB Nochlin, D Jin, LW Vincent, I
Citation: Jw. Husseman et al., Hyperphosphorylation of RNA polymerase II and reduced neuronal RNA levels precede neurofibrillary tangles in Alzheimer disease, J NE EXP NE, 60(12), 2001, pp. 1219-1232
Frequency of tau gene mutations in familial and sporadic cases of non-Alzheimer dementia
Authors: Poorkaj, P Grossman, M Steinbart, E Payami, H Sadovnick, A Nochlin, D Tabira, T Trojanowski, JQ Borson, S Galasko, D Reich, S Quinn, B Schellenberg, G Bird, TD
Citation: P. Poorkaj et al., Frequency of tau gene mutations in familial and sporadic cases of non-Alzheimer dementia, ARCH NEUROL, 58(3), 2001, pp. 383-387
Prion proteins with different conformations accumulate in Geustmann-Straussler-Scheinker disease caused by A117V and F198S mutations
Authors: Piccardo, P Liepnieks, JJ William, A Dlouhy, SR Farlow, MR Young, K Nochlin, D Bird, TD Nixon, RR Ball, MJ DeCarli, C Bugiani, O Tagliavini, F Benson, MD Ghetti, B
Citation: P. Piccardo et al., Prion proteins with different conformations accumulate in Geustmann-Straussler-Scheinker disease caused by A117V and F198S mutations, AM J PATH, 158(6), 2001, pp. 2201-2207
Brain expression of presenilins in sporadic and early-onset, familial Alzheimer's disease
Authors: Mathews, PM Cataldo, AM Kao, BH Rudnicki, AG Qin, X Yang, JL Jiang, Y Picciano, M Hulette, C Lippa, CF Bird, TD Nochlin, D Walter, J Haass, C Levesque, L Fraser, PE Andreadis, A Nixon, RA
Citation: Pm. Mathews et al., Brain expression of presenilins in sporadic and early-onset, familial Alzheimer's disease, MOL MED, 6(10), 2000, pp. 878-891
Familial Alzheimer's disease: Site of mutation influences clinical phenotype
Authors: Lippa, CF Swearer, JM Kane, KJ Nochlin, D Bird, TD Ghetti, B Nee, LE St George-Hyslop, P Pollen, DA Drachman, DA
Citation: Cf. Lippa et al., Familial Alzheimer's disease: Site of mutation influences clinical phenotype, ANN NEUROL, 48(3), 2000, pp. 376-379
Mitotic activation: a convergent mechanism for a cohort of neurodegenerative diseases
Authors: Husseman, JW Nochlin, D Vincent, I
Citation: Jw. Husseman et al., Mitotic activation: a convergent mechanism for a cohort of neurodegenerative diseases, NEUROBIOL A, 21(6), 2000, pp. 815-828
AMY plaques in familial AD - Comparison with sporadic Alzheimer's disease
Authors: Lippa, CF Schmidt, ML Nee, LE Bird, T Nochlin, D Hulette, C Mori, H Lee, VMY Trojanowski, JQ
Citation: Cf. Lippa et al., AMY plaques in familial AD - Comparison with sporadic Alzheimer's disease, NEUROLOGY, 54(1), 2000, pp. 100-104
The cell cycle Cdc25A tyrosine phosphatase is activated in degenerating postmitotic neurons in Alzheimer's disease
Authors: Ding, XL Husseman, J Tomashevski, A Nochlin, D Jin, LW Vincent, I
Citation: Xl. Ding et al., The cell cycle Cdc25A tyrosine phosphatase is activated in degenerating postmitotic neurons in Alzheimer's disease, AM J PATH, 157(6), 2000, pp. 1983-1990
The number of trait loci in late-onset Alzheimer disease
Authors: Daw, EW Payami, H Nemens, EJ Nochlin, D Bird, TD Schellenberg, GD Wijsman, EM
Citation: Ew. Daw et al., The number of trait loci in late-onset Alzheimer disease, AM J HU GEN, 66(1), 2000, pp. 196-204
Failure to detect Chlamydia pneumoniae in brain tissues of Alzheimer's disease
Authors: Nochlin, D Shaw, CM Campbell, LA Kuo, CC
Citation: D. Nochlin et al., Failure to detect Chlamydia pneumoniae in brain tissues of Alzheimer's disease, NEUROLOGY, 53(8), 1999, pp. 1888-1888
Missense and silent tau gene mutations cause frontotemporal dementia with parkinsonism-chromosome 17 type, by affecting multiple alternative RNA splicing regulatory elements
Authors: D'Souza, I Poorkaj, P Hong, M Nochlin, D Lee, VMY Bird, TD Schellenberg, GD
Citation: I. D'Souza et al., Missense and silent tau gene mutations cause frontotemporal dementia with parkinsonism-chromosome 17 type, by affecting multiple alternative RNA splicing regulatory elements, P NAS US, 96(10), 1999, pp. 5598-5603
The impact of different presenilin 1 and presenilin 2 mutations on amyloiddeposition, neurofibrillary changes and neuronal loss in the familial Alzheimer's disease brain - Evidence for other phenotype-modifying factors
Authors: Gomez-Isla, T Growdon, WB McNamara, MJ Nochlin, D Bird, TD Arango, JC Lopera, F Kosik, KS Lantos, PL Cairns, NJ Hyman, BT
Citation: T. Gomez-isla et al., The impact of different presenilin 1 and presenilin 2 mutations on amyloiddeposition, neurofibrillary changes and neuronal loss in the familial Alzheimer's disease brain - Evidence for other phenotype-modifying factors, BRAIN, 122, 1999, pp. 1709-1719
A clinical pathological comparison of three families with frontotemporal dementia and identical mutations in the tan gene (P301L) (vol 122, pg 741, 1999)
Authors: Bird, TD Nochlin, D Poorkaj, P Cherrier, M Payami, H Peskind, E Lampe, TH Nemens, E Boyer, PJ Schellenberg, GD
Citation: Td. Bird et al., A clinical pathological comparison of three families with frontotemporal dementia and identical mutations in the tan gene (P301L) (vol 122, pg 741, 1999), BRAIN, 122, 1999, pp. 1398-1398
A clinical pathological comparison of three families with frontotemporal dementia and identical mutations in the tau gene (P301L)
Authors: Bird, TD Nochlin, D Poorkaj, P Cherrier, M Kaye, J Payami, H Peskind, E Lampe, TH Nemens, E Boyer, PJ Schellenberg, GD
Citation: Td. Bird et al., A clinical pathological comparison of three families with frontotemporal dementia and identical mutations in the tau gene (P301L), BRAIN, 122, 1999, pp. 741-756
The utility of apolipoprotein E genotyping in the diagnosis of Alzheimer disease in a community-based case series
Authors: Tsuang, D Larson, EB Bowen, J McCormick, W Teri, L Nochlin, D Leverenz, JB Peskind, ER Lim, A Raskind, MA Thompson, ML Mirra, SS Gearing, M Schellenberg, GD Kukull, W
Citation: D. Tsuang et al., The utility of apolipoprotein E genotyping in the diagnosis of Alzheimer disease in a community-based case series, ARCH NEUROL, 56(12), 1999, pp. 1489-1495
Clinico-neuropathological correlation of Alzheimer's disease in a community-based case series
Authors: Lim, A Tsuang, D Kukull, W Nochlin, D Leverenz, J McCormick, W Bowen, J Teri, L Thompson, J Peskind, ER Raskind, M Larson, EB
Citation: A. Lim et al., Clinico-neuropathological correlation of Alzheimer's disease in a community-based case series, J AM GER SO, 47(5), 1999, pp. 564-569
Is neuropathological heterogeneity in Alzheimer's disease related to apolipoprotein genotype?
Authors: Armstrong, RA Wade, KM Nochlin, D Bird, TD
Citation: Ra. Armstrong et al., Is neuropathological heterogeneity in Alzheimer's disease related to apolipoprotein genotype?, ALZHEIM REP, 1(4), 1998, pp. 217-221
Transgenic mice over-expressing the C-99 fragment of beta PP with an alpha-secretase site mutation develop a myopathy similar to human inclusion bodymyositis
Authors: Jin, LW Hearn, MG Ogburn, CE Dang, N Nochlin, D Ladiges, WC Martin, GM
Citation: Lw. Jin et al., Transgenic mice over-expressing the C-99 fragment of beta PP with an alpha-secretase site mutation develop a myopathy similar to human inclusion bodymyositis, AM J PATH, 153(6), 1998, pp. 1679-1686
Risultati: 1-19 |