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Citation: Sh. Mead et al., A large family with hereditary spastic paraparesis due to a frame shift mutation of the spastin (SPG4) gene: association with multiple sclerosis in two affected siblings and epilepsy in other affected family members, J NE NE PSY, 71(6), 2001, pp. 788-791
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Citation: Pa. Brex et al., Measurement of spinal cord area in clinically isolated syndromes suggestive of multiple sclerosis, J NE NE PSY, 70(4), 2001, pp. 544-547
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Citation: Pa. Brex et al., Assessing the risk of early multiple sclerosis in patients with clinicallyisolated syndromes: the role of a follow up MRI, J NE NE PSY, 70(3), 2001, pp. 390-393
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Citation: Pa. Brex et al., Detection of ventricular enlargement in patients at the earliest clinical stage of MS, NEUROLOGY, 54(8), 2000, pp. 1689-1691
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Citation: Dj. Werring et al., Recovery from optic neuritis is associated with a change in the distribution of cerebral response to visual stimulation: a functional magnetic resonance imaging study, J NE NE PSY, 68(4), 2000, pp. 441-449
Citation: H. Manji et Gt. Plant, Epilepsy surgery, visual fields, and driving: a study of the visual field criteria for driving in patients after temporal lobe epilepsy surgery with a comparison of Goldmann and Esterman perimetry, J NE NE PSY, 68(1), 2000, pp. 80-82
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Citation: A. Brusa et al., Long-term recovery and fellow eye deterioration after optic neuritis, determined by serial visual evoked potentials, J NEUROL, 246(9), 1999, pp. 776-782
Authors:
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Citation: Bjl. Burton et al., Fibrous dysplasia of the sphenoid associated with visual loss and optic disc oedema, NEURO-OPHTH, 21(4), 1999, pp. 241-248
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Citation: M. Thom et al., Cortical dysplasia with angiodysgenesis and chronic inflammation in multifocal partial epilepsy, NEUROLOGY, 52(3), 1999, pp. 654-657